Practical precooling: Effect on cycling time trial performance in warm conditions

Abstract

The purpose of this study was to compare the effects of two practical precooling techniques (skin cooling vs. skin + core cooling) on cycling time trial performance in warm conditions. Six trained cyclists completed one maximal graded exercise test ([Vdot]O_2peak 71.4 ± 3.2 ml · kg^?1 · min^?1) and four ～40 min laboratory cycling time trials in a heat chamber (34.3°C ± 1.1°C; 41.2% ± 3.0% rh) using a fixed-power/variable-power format. Cyclists prepared for the time trial using three techniques administered in a randomised order prior to the warm-up: (1) no cooling (control), (2) cooling jacket for 40 min (jacket) or (3) 30-min water immersion followed by a cooling jacket application for 40 min (combined). Rectal temperature prior to the time trial was 37.8°C ± 0.1°C in control, similar in jacket (37.8°C ± 0.3°C) and lower in combined (37.1°C ± 0.2°C, P < 0.01). Compared with the control trial, time trial performance was not different for jacket precooling (?16 ± 36 s, ?0.7%; P = 0.35) but was faster for combined precooling (?42 ± 25 s, ?1.8%; P = 0.009). In conclusion, a practical combined precooling strategy that involves immersion in cool water followed by the use of a cooling jacket can produce decrease in rectal temperature that persist throughout a warm-up and improve laboratory cycling time trial performance in warm conditions.     

Metabolic cost of running is greater on a treadmill with a stiffer running platform

Exercise testing on motorised treadmills provides valuable information about running performance and metabolism; however, the impact of treadmill type on these tests has not been investigated. This study compared the energy demand of running on two laboratory treadmills: an HP Cosmos (C) and a Quinton (Q) model, with the latter having a 4.5 times stiffer running platform. Twelve experienced runners ran identical bouts on these treadmills at a range of four submaximal velocities (reported data is for the velocity that approximated 75–81% VO_2max). The stiffer treadmill elicited higher oxygen consumption (C: 46.7 ± 3.8; Q: 50.1 ± 4.3 ml·kg^?1 · min^?1), energy expenditure (C: 16.0 ± 2.5; Q: 17.7 ± 2.9 kcal · min^?1), carbohydrate oxidation (C: 9.6 ± 3.1; Q: 13.0 ± 3.9 kcal · min^?1), heart rate (C: 155 ± 16; Q: 163 ± 16 beats · min^?1) and rating of perceived exertion (C: 13.8 ± 1.2; Q: 14.7 ± 1.2), but lower fat oxidation (C: 6.4 ± 2.3; Q: 4.6 ± 2.5 kcal · min^?1) (all analysis of variance treadmill comparisons P < 0.01). This study confirms that caution is required when comparing performance and metabolic results between different treadmills and suggests that treadmills will vary in their comparability to over-ground running depending on the running platform stiffness.     

Dietary nitrate supplementation does not improve cycling time-trial performance in the heat

This investigation examined the effect of beetroot juice (BR) supplementation, a source of dietary nitrate (NO₃^?), on cycling time-trial (TT) performance and thermoregulation in the heat. In a double-blind, repeated-measures design, 12 male cyclists (age 26.6 ± 4.4 years, VO_2peak 65.8 ± 5.5 mL.kg^?1.min^?1) completed four cycling TTs (14 kJ.kg^?1) in hot (35°C, 48% relative humidity) and euthermic (21°C, 52%) conditions, following 3 days supplementation with BR (6.5 mmol NO₃^? for 2 days and 13 mmol NO₃^? on the final day), or NO₃^–depleted placebo (PLA). Salivary NO₃^? and nitrite, core (T_c) and mean skin temperature (T_sk) were measured. Salivary NO₃^? and nitrite increased significantly post-BR supplementation (p < 0.001). Average TT completion time (mm:ss) in hot conditions was 56:50 ± 05:08 with BR, compared with 58:30 ± 04:48 with PLA (p = 0.178). In euthermic conditions, average completion time was 53:09 ± 04:35 with BR, compared with 54:01 ± 04:05 with PLA (p = 0.380). The TT performance decreased (p < 0.001), and T_c (p < 0.001) and T_sk (p < 0.001) were higher in hot compared with euthermic conditions. In summary, BR supplementation has no significant effect on cycling TT performance in the heat.     

Similar results for face mask versus mouthpiece during incremental exercise to exhaustion

Investigations in the 1990s evaluated the influence of breathing assemblies on respiratory variables at rest and during exercise; however, research on new models of breathing assemblies is lacking. This study compared metabolic gas analysis data from a mouthpiece with a noseclip (MOUTH) and a face mask (MASK). Volunteers (7 males, 7 females; 25.1 ± 2.7 years) completed two maximal treadmill tests within 1 week, one MOUTH and one MASK, in random order. The difference in maximal oxygen consumption (VO_2max) between MOUTH (52.7 ± 11.3 ml · kg^?1 · min^?1) and MASK (52.2 ± 11.7 ml · kg^?1 · min^?1) was not significant (P = 0.53). Likewise, the mean MOUTH–MASK differences in minute ventilation (V_E), fraction of expired oxygen (FEO₂) and carbon dioxide (FECO₂), respiration rate (RR), tidal volume (V_t), heart rate (HR), and rating of perceived exertion (RPE) at maximal and submaximal intensities were not significant (P > 0.05). Furthermore, there was no systematic bias in the error scores (r = ?0.13, P = 0.66), and 12 of the 14 participants had a VO_2max difference of ≤3 ml · kg^?1 · min^?1 between conditions. Finally, there was no clear participant preference for using the MOUTH or MASK. Selection of MOUTH or MASK will not affect the participant’s gas exchange or breathing patterns.     

Monitoring changes in VO2max via the Polar FT40 in female collegiate soccer players

Abstract

This study was conducted to determine if the Polar FT40 could accurately track changes in maximal oxygen consumption (VO_2max) in a group of female soccer players. Predicted VO_2max (pVO_2max) via the Polar FT40 and observed VO_2max (aVO_2max) from a maximal exercise test on a treadmill were determined for members of a collegiate soccer team (n = 20) before and following an 8-week endurance training protocol. Predicted (VO_2max and aVO_2max measures were compared at baseline and within 1 week post-training. Change values (i.e., the difference between pre to post) for each variable were also determined and compared. There was a significant difference in aVO_2max (pre = 43.6 ± 2.4 ml · kg · min^?1, post = 46.2 ± 2.4 ml · kg · min^?1, P < 0.001) and pVO_2max (pre = 47.3 ± 5.3 ml · kg · min^?1, post = 49.7 ± 6.2 ml · kg · min^?1, P = 0.009) following training. However, predicted values were significantly greater at each time point compared to observed values (P < 0.001 at pre and P = 0.008 at post). Furthermore, there was a weak correlation between the change in aVO_2max and the change in pVO_2max (r = 0.18, P = 0.45). The Polar FT40 does not appear to be a valid method for predicting changes in individual VO_2max following 8 weeks of endurance training in female collegiate soccer players.     

Specificity of a Maximal Step Exercise Test

To adhere to the principle of “exercise specificity” exercise testing should be completed using the same physical activity that is performed during exercise training. The present study was designed to assess whether aerobic step exercisers have a greater maximal oxygen consumption (max VO₂) when tested using an activity specific, maximal step exercise test (SET; arms and legs) versus a maximal running test (legs only). Female aerobic step exercisers (N=18; 20.7 ± 1.5 years) performed three maximal graded exercise tests (GXTs): 2 SETs; 1 treadmill test (TMT). The SET consisted of six 3-min progressive stages of alternate lead, basic step, basic step with biceps curls, knee raise with pull-down, repeater knee with pull-down, lateral lunge with pull-down, and side squat with shoulder presses. Stepping rate was 32 steps· min^?1 on an 8-in (20.32 cm) step for stages 1–3, and a 10-in (25.4 cm) step for stages 4–6. Submaximal and maximal heart rate (HR) and oxygen consumption (VO₂) were recorded at the end of each stage. Test–retest reliability for the first five stages of the SET ranged from .91 to .97 for HR, and from .84 to .96 for VO₂. Maximal HR was significantly greater (p =.0001) for the SET (200 ± 6.2 beats·min^?1) as compared to the TMT (193 ± 7.9 beats·min^?1). No significant difference was found for max VO₂ (42.9 ± 8.5, 41.2 ± 5.9 ml·kg^?1·min^?1, p =.14). The SET was a valid and reliable protocol for assessing responses of these aerobic step exercisers; however, max VO₂ from a TMT did not differ significantly from the SET. Conversely, max HR obtained from the criterion TMT was 7 beats·min^?1 lower than from the SET. If a training HR for step exercise (arms and legs exercise) is prescribed based on the max HR from treadmill exercise (legs only), then the training HR should be calculated from a TMT max HR that has been increased by 7 beats·min^?1 to obtain an intensity of step exercise comparable to that of running.     

Effects of high-intensity interval training on cardiometabolic risk factors in overweight/obese women

The purpose of this study was to evaluate two practical interval training protocols on cardiorespiratory fitness, lipids and body composition in overweight/obese women. Thirty women (mean ± SD; weight: 88.1 ± 15.9 kg; BMI: 32.0 ± 6.0 kg · m²) were randomly assigned to ten 1-min high-intensity intervals (90%VO₂ peak, 1 min recovery) or five 2-min high-intensity intervals (80–100% VO₂ peak, 1 min recovery) or control. Peak oxygen uptake (VO₂ peak), peak power output (PPO), body composition and fasting blood lipids were evaluated before and after 3 weeks of training, completed 3 days per week. Results from ANCOVA analyses demonstrated no significant training group differences for any primary variables (P > 0.05). When training groups were collapsed, 1MIN and 2MIN resulted in a significant increase in PPO (?18.9 ± 8.5 watts; P = 0.014) and time to exhaustion (?55.1 ± 16.4 s; P = 0.001); non-significant increase in VO₂ peak (?2.36 ± 1.34 ml · kg^?1 · min^?1; P = 0.185); and a significant decrease in fat mass (FM) (??1.96 ± 0.99 kg; P = 0.011). Short-term interval exercise training may be effective for decreasing FM and improving exercise tolerance in overweight and obese women.     

Submaximal Treadmill Exercise Test to Predict VO2max in Fit Adults

This study was designed to develop a single-stage submaximal treadmill jogging (TMJ) test to predict VO₂max in fit adults. Participants (N?=?400; men?=?250 and women?=?150), ages 18 to 40 years, successfully completed a maximal graded exercise test (GXT) at 1 of 3 laboratories to determine VO₂max. The TMJ test was completed during the first 2 stages of the GXT. Following 3 min of walking (Stage 1), participants achieved a steady-state heart rate (HR) while exercising at a comfortable self-selected submaximal jogging speed at level grade (Stage 2). Gender, age, body mass, steady-state HR, and jogging speed (mph) were included as independent variables in the following multiple linear regression model to predict VO₂max (R?=?0.91, standard error of estimate [SEE]?=?2.52 mL?·?kg^?1?·?min^?1): VO₂max (mL?·?kg^?1?·?min^?1)?=?58.687?+?(7.520 × Gender; 0?=?woman and 1?=?man)?+?(4.334 × mph) ? (0.211 × kg) ? (0.148 × HR) ? (0.107 × Age). Based on the predicted residual sum of squares (PRESS) statistics (R_PRESS?=?0.91, SEE _PRESS?=?2.54 mL?·?kg^?1?·?min^?1) and small total error (TE; 2.50 mL?·?kg^?1?·?min^?1; 5.3% of VO₂max) and constant error (CE; ?0.008 mL?·?kg^?1?·?min^?1) terms, this new prediction equation displays minimal shrinkage. It should also demonstrate similar accuracy when it is applied to other samples that include participants of comparable age, body mass, and aerobic fitness level. This simple TMJ test and its corresponding regression model provides a relatively safe, convenient, and accurate way to predict VO₂max in fit adults, ages 18 to 40 years.     

Combined effects of exposure to hypoxia and cool on walking economy and muscle oxygenation profiles at tibialis anterior

We investigated combined effects of ambient temperature (23°C or 13°C) and fraction of inspired oxygen (21%O₂ or 13%O₂) on energy cost of walking (C_w: J·kg^?1·km^?1) and economical speed (ES). Eighteen healthy young adults (11 males, seven females) walked at seven speeds from 0.67 to 1.67 m s^?1 (four min per stage). Environmental conditions were set; thermoneutral (N: 23°C) with normoxia (N: 21%O₂) = NN; 23°C (N) with hypoxia (H: 13%O₂) = NH; cool (C: 13°C) with 21%O₂ (N) = CN, and 13°C (C) with 13%O₂ (H) = CH. Muscle deoxygenation (HHb) and tissue O₂ saturation (StO₂) were measured at tibialis anterior. We found a significantly slower ES in NH (1.289 ± 0.091 m s^?1) and CH (1.275 ± 0.099 m s^?1) than in NN (1.334 ± 0.112 m s^?1) and CN (1.332 ± 0.104 m s^?1). Changes in HHb and StO₂ were related to the ES. These results suggested that the combined effects (exposure to hypoxia and cool) is nearly equal to exposure to hypoxia and cool individually. Specifically, acute moderate hypoxia slowed the ES by approx. 4%, but acute cool environment did not affect the ES. Further, HHb and StO₂ may partly account for an individual ES.     

Hydration,sweat and thermoregulatory responses to professional football training in the heat

Abstract

This study examined the relationship between intensity of training and changes in hydration status, core temperature, sweat rate and composition and fluid balance in professional football players training in the heat. Thirteen professional football players completed three training sessions; “higher-intensity” (140 min; HI₁₄₀), “lower-intensity” (120 min; LI₁₂₀) and “game-simulation” (100 min; GS₁₀₀). Movement demands were measured by Global Positioning System, sweat rate and concentration were determined from dermal patches and body mass change. Despite similar environmental conditions (26.9 ± 0.1°C and 65.0 ± 7.0% relative humidity [Rh]), higher relative speeds (m · min^?1) and increased perceptions of effort and thermal strain were observed in HI₁₄₀ and GS₁₀₀ compared with LI₁₂₀ (P < 0.05). Significantly (P < 0.05) greater sweat rate (L · h^?1) and electrolyte losses (g) were observed in HI₁₄₀ and GS₁₀₀ compared with LI₁₂₀. Rate of rise in core temperature was correlated with mean speed (r = 0.85), session rating of perceived exertion (sRPE) (r = 0.61), loss of potassium (K⁺) (r = 0.51) sweat rate (r = 0.49), and total sweat loss (r = 0.53), with mean speed the strongest predictor. Sodium (Na⁺) (r = 0.39) and K⁺ (r = 0.50) losses were associated with total distance covered. In hot conditions, individualised rehydration practices should be adopted following football training to account for differences in sweat rate and electrolyte losses in response to intensity and overall activity within a session.     

Carbohydrate intake and training efficacy – a randomized cross-over study

Carbohydrate (CHO) availability during endurance exercise seems to attenuate exercise-induced perturbations of cellular homeostasis and might consequently diminish the stimulus for training adaptation. Therefore, a negative effect of CHO intake on endurance training efficacy seems plausible. This study aimed to test the influence of carbohydrate intake on the efficacy of an endurance training program on previously untrained healthy adults. A randomized cross-over trial (8-week wash-out period) was conducted in 23 men and women with two 8-week training periods (with vs. without intake of 50g glucose before each training bout). Training intervention consisted of 4x45 min running/walking sessions/week at 70% of heart rate reserve. Exhaustive, ramp-shaped exercise tests with gas exchange measurements were conducted before and after each training period. Outcome measures were maximum oxygen uptake (VO_2max) and ventilatory anaerobic threshold (VT). VO_2max and VT increased after training regardless of CHO intake (VO_2max: Non-CHO 2.6 ± 3.0 ml*min^?1*kg^?1 p = 0.004; CHO 1.4 ± 2.5 ml*min^?1*kg^?1 p = 0.049; VT: Non-CHO 4.2 ± 4.2 ml*min^?1*kg^?1 p < 0.001; CHO 3.0 ± 4.2 ml*min^?1*kg^?1 p = 0.003). The 95% confidence interval (CI) for the difference between conditions was between +0.1 and +2.1 ml*min^?1*kg^?1 for VO_2max and between ?1.2 and +3.1 for VT. It is concluded that carbohydrate intake could potentially impair the efficacy of an endurance training program.     

Effect of biological maturation on maximal oxygen uptake and ventilatory thresholds in soccer players: An allometric approach

Abstract

In this study, we investigated the effect of biological maturation on maximal oxygen uptake ([Vdot]O_2max) and ventilatory thresholds (VT₁ and VT₂) in 110 young soccer players separated into pubescent and post-pubescent groups.. Maximal oxygen uptake and [Vdot]O₂ corresponding to VT₁ and VT₂ were expressed as absolute values, ratio standards, theoretical exponents, and experimentally observed exponents. Absolute [Vdot]O₂ (ml · min^?1) was different between groups for VT₁, VT₂, and [Vdot]O_2max. Ratio standards (ml · kg^?1 · min^?1) were not significantly different between groups for VT₁, VT₂, and [Vdot]O_2max. Theoretical exponents (ml · kg^?0.67 · min^?1 and ml · kg^?0.75 · min^?1) were not properly adjusted for the body mass effects on VT₁, VT₂, and [Vdot]O_2max. When the data were correctly adjusted using experimentally observed exponents, VT₁ (ml · kg^?0.94 · min^?1) and VT₂ (ml · kg^?0.95 · min^?1) were not different between groups. The experimentally observed exponent for [Vdot]O_2max (ml · kg^?0.90 · min^?1) was different between groups (P = 0.048); however, this difference could not be attributed to biological maturation. In conclusion, biological maturation had no effect on VT₁, VT₂ or [Vdot]O_2max when the effect of body mass was adjusted by experimentally observed exponents. Thus, when evaluating the physiological performance of young soccer players, allometric scaling needs to be taken into account instead of using theoretical approaches.     

Understanding mental toughness in Australian soccer: Perceptions of players,parents, and coaches

Abstract

This study was designed to investigate the effect of ingesting a glucose plus fructose solution on the metabolic responses to soccer-specific exercise in the heat and the impact on subsequent exercise capacity. Eleven male soccer players performed a 90 min soccer-specific protocol on three occasions. Either 3 ml · kg^?1 body mass of a solution containing glucose (1 g · min^?1 glucose) (GLU), or glucose (0.66 g · min^?1) plus fructose (0.33 g · min^?1) (MIX) or placebo (PLA) was consumed every 15 minutes. Respiratory measures were undertaken at 15-min intervals, blood samples were drawn at rest, half-time and on completion of the protocol, and muscle glycogen concentration was assessed pre- and post-exercise. Following the soccer-specific protocol the Cunningham and Faulkner test was performed. No significant differences in post-exercise muscle glycogen concentration (PLA, 62.99 ± 8.39 mmol · kg wet weight^?1; GLU 68.62 ± 2.70; mmol · kg wet weight^?1 and MIX 76.63 ± 6.92 mmol · kg wet weight^?1) or exercise capacity (PLA, 73.62 ± 8.61 s; GLU, 77.11 ± 7.17 s; MIX, 83.04 ± 9.65 s) were observed between treatments (P > 0.05). However, total carbohydrate oxidation was significantly increased during MIX compared with PLA (P < 0.05). These results suggest that when ingested in moderate amounts, the type of carbohydrate does not influence metabolism during soccer-specific intermittent exercise or affect performance capacity after exercise in the heat.     

The influence of serial feeding of drinks at different temperatures on thermoregulatory responses during cycling

Abstract

In this study, we examined thermoregulatory responses to ingestion of separate aliquots of drinks at different temperatures during low-intensity exercise in conditions of moderate heat stress. Eight men cycled at 50% (s = 3) of their peak oxygen uptake ([Vdot]O_2peak) for 90 min (dry bulb temperature: 25.3°C, s = 0.5; relative humidity: 60%, s = 5). Four 400-ml aliquots of flavoured water at 10°C (cold), 37°C (warm) or 50°C (hot) were ingested after 30, 45, 60, and 75 min of exercise. Immediately after the 90 min of exercise, participants cycled at 95%[Vdot]O_2peak to exhaustion to assess exercise capacity. There were no differences between trials in rectal temperature at the end of the 90 min of exercise (cold: 38.11°C, s = 0.30; warm: 38.10°C, s = 0.33; hot: 38.21°C, s = 0.30; P = 0.765). Mean skin temperature between 30 and 90 min tended to be influenced by drink temperature (cold: 34.49°C, s = 0.64; warm: 34.53°C, s = 0.69; hot: 34.71°C, s = 0.48; P = 0.091). Mean heart rate from 30 to 90 min was higher in the hot trial (129 beats · min^?1, s = 7; P < 0.05) than on the cold (124 beats · min^?1, s = 9) and warm trials (126 beats · min^?1, s = 8). Ratings of thermal sensation were higher on the hot trial than on the cold trial at 35 and 50 min (P < 0.05). Exercise capacity was similar between trials (P = 0.963). The heat load and debt induced by periodic drinking resulted in similar body temperatures during low-intensity exercise in conditions of moderate heat stress due to appropriate thermoregulatory reflexes.     

Low volume–high intensity interval exercise elicits antioxidant and anti-inflammatory effects in humans

The purpose of the present study was to compare acute changes in oxidative stress and inflammation in response to steady state and low volume, high intensity interval exercise (LV-HIIE). Untrained healthy males (n = 10, mean ± s: age 22 ± 3 years; VO_2MAX 42.7 ± 5.0 ml · kg^?1 · min^?1) undertook three exercise bouts: a bout of LV-HIIE (10 × 1 min 90% VO_2MAX intervals) and two energy-matched steady-state cycling bouts at a moderate (60% VO_2MAX; 27 min, MOD) and high (80% VO_2MAX; 20 min, HIGH) intensity on separate days. Markers of oxidative stress, inflammation and physiological stress were assessed before, at the end of exercise and 30 min post-exercise (post+30). At the end of all exercise bouts, significant changes in lipid hydroperoxides (LOOH) and protein carbonyls (PCs) (LOOH (nM): MOD +0.36; HIGH +3.09; LV-HIIE +5.51 and PC (nmol · mg^?1 protein): MOD ?0.24; HIGH ?0.11; LV-HIIE ?0.37) were observed. Total antioxidant capacity (TAC) increased post+30, relative to the end of all exercise bouts (TAC (µM): MOD +189; HIGH +135; LV-HIIE +102). Interleukin (IL)-6 and IL-10 increased post+30 in HIGH and LV-HIIE only (P < 0.05). HIGH caused the greatest lymphocytosis, adrenaline and cardiovascular response (P < 0.05). At a reduced energy cost and physiological stress, LV-HIIE elicited similar cytokine and oxidative stress responses to HIGH.     

Development of 1-mile walk tests to estimate aerobic fitness in children

To examine the reliability and validity of 1-mile walk tests for estimation of aerobic fitness (VO_2max) in 10- to 13-year-old children and to cross-validate previously published equations. Participants (n = 61) walked 1-mile on two different days. Self-reported physical activity, demographic variables, and aerobic fitness were used in multiple regression analyses. Eight models were developed with various combinations of predictors. The recommended model for fitness testing in schools was: VO_2max = 120.702 + (4.114 × Sex [F = 0, M = 1]) – (2.918 × 1-mile Walk Time [min]) – (2.841 × Age), R = .73, standard error of estimate = 6.36 mL·kg^?1·min^?1. Cross-validation of previously published equations demonstrated lower correlations with measured VO_2max than the newly developed walk tests. Evidence of reliability and validity for 1-mile walk tests to estimate VO_2max in young children was provided. The model that included 1-mile walk time, age, and sex may be appropriate for youth fitness testing in physical education, particularly for unmotivated or overweight young children.     

A Maximal Graded Exercise Test to Accurately Predict VO2max in 18–65-Year-Old Adults

The purpose of this study was to develop an age-generalized regression model to predict maximal oxygen uptake (VO_2max) based on a maximal treadmill graded exercise test (GXT; George, 1996) George, J. D. 1996. Alternative approach to maximal exercise testing and VO_2max prediction in college students. Research Quarterly for Exercise and Sport, 67: 452–457. [Taylor & Francis Online], [Web of Science ®] , [Google Scholar]. Participants (N?=?100), ages 18–65 years, reached a maximal level of exertion (mean?±?standard deviation [SD]; maximal heart rate [HR_max]?=?185.2?±?12.4 beats per minute (bpm); maximal respiratory exchange ratio [RER_max]?=?1.18?±?0.05; maximal rating of perceived exertion (RPE_max)?=?19.1?±?0.7) during the GXT to assess VO_2max (mean?±?SD; 40.24?±?9.11 mL·kg^?1·min^?1). Multiple linear regression generated the following prediction equation (R?=?.94, standard error of estimate [SEE]?=?3.18 mL·kg^?1·min^?1, %SEE?=?7.9): VO_2max (mL·kg^?1·min^?1)?=?13.160?+?(3.314 × gender; females?=?0, males?=?1) ? (.131 × age) ? (.334 × body mass index (BMI))?+?(5.177 × treadmill speed; mph)?+?(1.315 × treadmill grade; %). Cross validation using predicted residual sum of squares (PRESS) statistics revealed minimal shrinkage (R_p ?=?.93 and SEE _p ?=?3.40 mL·kg^?1·min^?1); consequently, this model should provide acceptable accuracy when it is applied to independent samples of comparable adults. Standardized β-weights indicate that treadmill speed (.583) was the most effective at predicting VO_2max followed by treadmill grade (.356), age (?.197), gender (.183), and BMI (?.148). This study provides a relatively accurate regression model to predict VO_2max in relatively fit men and women, ages 18–65 years, based on maximal exercise (treadmill speed and grade), biometric (BMI), and demographic (age and gender) data.     

Rain influences the physiological and metabolic responses to exercise in hot conditions

Outdoor exercise often proceeds in rainy conditions. However, the cooling effects of rain on human physiological responses have not been systematically studied in hot conditions. The present study determined physiological and metabolic responses using a climatic chamber that can precisely simulate hot, rainy conditions. Eleven healthy men ran on a treadmill at an intensity of 70% VO_2max for 30 min in the climatic chamber at an ambient temperature of 33°C in the presence (RAIN) or absence (CON) of 30 mm · h^?1 of precipitation and a headwind equal to the running velocity of 3.15 ± 0.19 m · s^?1. Oesophageal temperature, mean skin temperature, heart rate, rating of perceived exertion, blood parameters, volume of expired air and sweat loss were measured. Oesophageal and mean skin temperatures were significantly lower from 5 to 30 min, and heart rate was significantly lower from 20 to 30 min in RAIN than in CON (P < 0.05 for all). Plasma lactate and epinephrine concentrations (30 min) and sweat loss were significantly lower (P < 0.05) in RAIN compared with CON. Rain appears to influence physiological and metabolic responses to exercise in heat such that heat-induced strain might be reduced.     

Maximal oxygen uptake versus maximal power output in children

Abstract

Maximal oxygen uptake ([Vdot]O_2max) is considered the optimal method to assess aerobic fitness. The measurement of [Vdot]O_2max, however, requires special equipment and training. Maximal exercise testing with determination of maximal power output offers a more simple approach. This study explores the relationship between [Vdot]O_2max and maximal power output in 247 children (139 boys and 108 girls) aged 7.9–11.1 years. Maximal oxygen uptake was measured by indirect calorimetry during a maximal ergometer exercise test with an initial workload of 30 W and 15 W · min^?1 increments. Maximal power output was also measured. A sample (n = 124) was used to calculate reference equations, which were then validated using another sample (n = 123). The linear reference equation for both sexes combined was: [Vdot]O_2max (ml · min^?1) = 96 + 10.6 · maximal power + 3.5 · body mass. Using this reference equation, estimated [Vdot]O_2max per unit of body mass (ml · min^?1 · kg^?1) calculated from maximal power correlated closely with the direct measurement of [Vdot]O_2max (r = 0.91, P <0.001). Bland-Altman analysis gave a mean limits of agreement of 0.2±2.9 (ml · min^?1 · kg^?1) (1 s). Our results suggest that maximal power output serves as a good surrogate measurement for [Vdot]O_2max in population studies of children aged 8–11 years.     

Low energy availability in exercising men is associated with reduced leptin and insulin but not with changes in other metabolic hormones

Low energy availability, defined as low caloric intake relative to exercise energy expenditure, has been linked to endocrine alterations frequently observed in chronically energy-deficient exercising women. Our goal was to determine the endocrine effects of low energy availability in exercising men. Six exercising men (VO_2peak: 49.3 ± 2.4 ml · kg^?1 · min^?1) underwent two conditions of low energy availability (15 kcal · kg^?1 fat-free mass [FFM] · day^?1) and two energy-balanced conditions (40 kcal · kg^?1 FFM · day^?1) in randomised order. During one low energy availability and one balanced condition, participants exercised to expend 15 kcal · kg^?1 FFM · day^?1; no exercise was conducted during the other two conditions. Metabolic hormones were assessed before and after each 4-day period. Following both low energy availability conditions, leptin (?53% to ?56%) and insulin (?34% to ?38%) were reduced (P < 0.05). Reductions in leptin and insulin were independent of whether low energy availability was attained with or without exercise (P > 0.80). Low energy availability did not significantly impact ghrelin, triiodothyronine, testosterone and IGF-1 (all P > 0.05). The observed reductions in leptin and insulin were in the same magnitude as changes previously reported in sedentary women. Further research is needed to understand why other metabolic hormones are more robust against low energy availability in exercising men than those in sedentary and exercising women.