Effects of work-matched supramaximal intermittent vs. submaximal constant-workload warm-up on all-out effort power output at the end of 2 minutes of maximal cycling

We tested the hypothesis that work-matched supramaximal intermittent warm-up improves final-sprint power output to a greater degree than submaximal constant-intensity warm-up during the last 30?s of a 120-s supramaximal exercise simulating the final sprint during sports events lasting approximately 2?min. Ten male middle-distance runners performed a 120-s supramaximal cycling exercise consisting of 90?s of constant-workload cycling at a workload corresponding to 110% maximal oxygen uptake (VO_2max) followed by 30?s of maximal-effort cycling. This exercise was preceded by 1) no warm-up (Control), 2) a constant-workload cycling warm-up at a workload of 60%VO_2max for 6?min and 40?s, or 3) a supramaximal intermittent cycling warm-up for 6?min and 40?s consisting of 5 sets of 65?s of cycling at a workload of 46%VO_2max?+?15?s of supramaximal cycling at a workload of 120%VO_2max. By design, total work was matched between the two warm-up conditions. Supramaximal intermittent and submaximal constant-workload warm-ups similarly increased 5-s peak (590?±?191 vs. 604?±?215W, P?=?0.41) and 30-s mean (495?±?137 vs. 503?±?154W, P?=?0.48) power output during the final 30-s maximal-effort cycling as compared to the no warm-up condition (5-s peak: 471?±?165W; 30-s mean: 398?±?117W). VO₂ during the 120-s supramaximal cycling was similarly increased by the two warm-ups as compared to no-warm up (P?≤?0.05). These findings show that work-matched supramaximal intermittent and submaximal constant-workload warm-ups improve final sprint (～30?s) performance to similar extents during the late stage of a 120-s supramaximal exercise bout.     

3-min all-out effort on cycle ergometer is valid to estimate the anaerobic capacity by measurement of blood lactate and excess post-exercise oxygen consumption

The purpose of this study was to investigate the use of a single 3-min all-out maximal effort to estimate anaerobic capacity (AC) through the lactate and excess post-exercise oxygen consumption (EPOC) response methods (AC_{[La^?]+EPOCfast}) on a cycle ergometer. Eleven physically active men (age?=?28.1?±?4.0?yrs, height?=?175.1?±?4.2?cm, body mass?=?74.8?±?11.9?kg and ?O_2max?=?40.7?±?7.3?mL?kg^?1?min^?1), participated in the study and performed: i) five submaximal efforts, ii) a supramaximal effort at 115% of intensity of ?O_2max, and iii) a 3-min all-out maximal effort. Anaerobic capacity was estimated using the supramaximal effort through conventional maximal accumulated oxygen deficit (MAOD) and also through the sum of oxygen equivalents from the glycolytic (fast component of excess post-exercise oxygen consumption) and phosphagen pathways (blood lactate accumulation) (AC_{[La^?]+EPOCfast}), while during the 3-min all-out maximal effort the anaerobic capacity was estimated using the AC_{[La^?]+EPOCfast} procedure. There were no significant differences between the three methods (p?>?0.05). Additionally, the anaerobic capacity estimated during the 3-min all-out effort was significantly correlated with the MAOD (r?=?0.74; p?=?0.009) and AC_{[La^?]+EPOCfast} methods (r?=?0.65; p?=?0.029). Therefore, it is possible to conclude that the 3-min all-out effort is valid to estimate anaerobic capacity in physically active men during a single cycle ergometer effort.     

Skeletal muscle glycogen concentration and metabolic responses following a high glycaemic carbohydrate breakfast

The purpose of this study was to examine the influence of a carbohydrate-rich meal on post-prandial metabolic responses and skeletal muscle glycogen concentration. After an overnight fast, eight male recreational/club endurance runners ingested a carbohydrate (CHO) meal (2.5 g CHO?·?kg^?1 body mass) and biopsies were obtained from the vastus lateralis muscle before and 3 h after the meal. Ingestion of the meal resulted in a 10.6?±?2.5% (P?<?0.05) increase in muscle glycogen concentration (pre-meal vs post-meal: 314.0?±?33.9 vs 347.3?±?31.3 mmol?·?kg^?1 dry weight). Three hours after ingestion, mean serum insulin concentrations had not returned to pre-feeding values (0 min vs 180 min: 45?±?4 vs 143?±?21 pmol?·?l^?1). On a separate occasion, six similar individuals ingested the meal or fasted for a further 3 h during which time expired air samples were collected to estimate the amount of carbohydrate oxidized over the 3 h post-prandial period. It was estimated that about 20% of the carbohydrate consumed was converted into muscle glycogen, and about 12 % was oxidized. We conclude that a meal providing 2.5 g CHO?·?kg^?1 body mass can increase muscle glycogen stores 3 h after ingestion. However, an estimated 67% of the carbohydrate ingested was unaccounted for and this may have been stored as liver glycogen and/or still be in the gastrointestinal tract.     

Contextual factors on physical demands in professional women’s soccer: Female Athletes in Motion study

The aim of this study was to examine the impact of contextual factors on relative locomotor and metabolic power distances during professional female soccer matches. Twenty-eight players (forwards, n?=?4; midfielders, n?=?12; defenders, n?=?12) that competed in a 90-min home and away match (regular season only). The generalised estimating equations (GEE) was used to evaluate relative locomotor and metabolic power distances for three contextual factors: location (home vs. away), type of turf (natural vs. artificial), and match outcome (win, loss and draw). No differences were observed for home vs. away matches. Moderate-intensity running (20.0?±?1.0?m?min^?1 and 16.4?±?0.9?m?min^?1), high-intensity running (8.6?±?0.4?m?min^?1 and 7.3?±?0.4?m?min^?1) and high-metabolic power (16.3?±?0.5?m?min^?1 and 14.4?±?0.5?m?min^?1) distances were elevated on artificial turf compared to natural grass, respectively. Relative sprint distance was greater during losses compared with draws (4.3?±?0.4?m?min^?1 and 3.4?±?0.3?m?min^?1). Overall physical demands of professional women’s soccer were not impacted by match location. However, the elevation of moderate and high-intensity demands while playing on artificial turf may have implications on match preparations as well as recovery strategies.     

Quantifying the effects of acute hypoxic exposure on exercise performance and capacity: A systematic review and meta-regression

Objective: To quantify the effects of acute hypoxic exposure on exercise capacity and performance, which includes continuous and intermittent forms of exercise. Design: A systematic review was conducted with a three-level mixed effects meta-regression. The ratio of means method was used to evaluate main effects and moderators providing practical interpretations with percentage change. Data sources: A systemic search was performed using three databases (Google scholar, PubMed and SPORTDiscus). Eligibility criteria for selecting studies: Inclusion was restricted to investigations that assessed exercise performance (time trials (TTs), sprint and intermittent exercise tests) and capacity (time to exhaustion test, TTE) with acute hypoxic (<24?h) exposure and a normoxic comparator. Results: Eighty-two outcomes from 53 studies (N?=?798) were included in this review. The results show an overall reduction in exercise performance/capacity ?17.8?±?3.9% (95% CI ?22.8% to ?11.0%), which was significantly moderated by ?6.5?±?0.9% per 1000 m altitude elevation (95% CI ?8.2% to ?4.8%) and oxygen saturation (?2.0?±?0.4%; 95% CI ?2.9% to ?1.2%). TT (?16.2?±?4.3%; 95% CI ?22.9% to ?9%) and TTE (?44.5?±?6.9%; 95% CI ?51.3% to ?36.7%) elicited a negative effect, whilst indicating a quadratic relationship between hypoxic magnitude and both TTE and TT performance. Furthermore, exercise less than 2 min exhibited no ergolytic effect from acute hypoxia.

Summary/Conclusion: This review highlights the ergolytic effect of acute hypoxic exposure, which is curvilinear for TTE and TT performance with increasing hypoxic levels, but short duration intermittent and sprint exercise seem to be unaffected.     

Influence of a caffeine mouth rinse on sprint cycling following glycogen depletion

Attenuated performance during intense exercise with limited endogenous carbohydrate (CHO) is well documented. Therefore, this study examined whether caffeine (CAF) mouth rinsing would augment performance during repeated sprint cycling in participants with reduced endogenous CHO. Eight recreationally active males (aged 23?±?2?yr, body mass 84?±?4?kg, stature 178?±?7?cm) participated in this randomized, single-blind, repeated-measures crossover investigation. Following familiarization, participants attended two separate evening glycogen depletion sessions. The following morning, participants completed five, 6?s sprints on a cycle ergometer (separated by 24?s active recovery), with mouth rinsing either (1) a placebo solution or (2) a 2% CAF solution. During a fifth visit, participants completed the sprints without prior glycogen depletion. Repeated-measures ANOVA identified significant main effect of condition (CAF, placebo, and control [P?P?P?P?P?P?相似文献   

Emotional pictures impact repetitive sprint ability test on cycle ergometre

Abstract

This study investigated the interaction between emotion-eliciting pictures and power output during a repetitive supra-maximal task on a cycle ergometre. Twelve male participants (mean (±SD) age, height and weight: 28.58 ± 3.23 years, 1.78 ± 0.05 m and 82.41 ± 13.29 kg) performed 5 repeated sprint tests on a cycle ergometre in front of neutral, pleasant or unpleasant pictures. For each sprint, mechanical (peak power and work), physiological (heart rate) and perceptual (affective load) indices were analysed. Affective load was calculated from the ratings of perceived exertion, which reflected the amount of pleasant and unpleasant responses experienced during exercise. The results showed that peak power, work and heart rate values were significantly lower (P < 0.05) for unpleasant pictures (9.18 ± 0.20 W ? kg^?1; 47.69 ± 1.08 J ? kg^?1; 152 ± 4 bpm) when compared with pleasant ones (9.50 ± 0.20 W ? kg^?1; 50.11 ± 0.11 J ? kg^?1; 156 ± 3 bpm). Furthermore, the affective load was found to be similar for the pleasant and unpleasant sessions. All together, these results suggested that the ability to produce maximal power output depended on whether the emotional context was pleasant or unpleasant. The fact that the power output was lower in the unpleasant versus pleasant session could reflect a regulatory process aimed at maintaining a similar level of affective load for both sessions.     

Normobaric hypoxia increases the growth hormone response to maximal resistance exercise in trained men

This study examined the effect of hypoxia on growth hormone (GH) release during an acute bout of high-intensity, low-volume resistance exercise. Using a single-blinded, randomised crossover design, 16 resistance-trained males completed two resistance exercise sessions in normobaric hypoxia (HYP; inspiratory oxygen fraction, (FiO₂) 0.12, arterial oxygen saturation (SpO₂) 82?±?2%) and normoxia (NOR; FiO₂ 0.21, SpO₂ 98?±?0%). Each session consisted of five sets of three repetitions of 45° leg press and bench press at 85% of one repetition maximum. Heart rate, SpO₂, and electromyographic activity (EMG) of the vastus lateralis muscle were measured throughout the protocol. Serum lactate and GH levels were determined pre-exposure, and at 5, 15, 30 and 60?min post-exercise. Differences in mean and integrated EMG between HYP and NOR treatments were unclear. However, there was an important increase in the peak levels and area under the curve of both lactate (HYP 5.8?±?1.8 v NOR 3.9?±?1.1?mmol.L^?1 and HYP 138.7?±?33.1 v NOR 105.8?±?20.8?min.mmol.L^?1) and GH (HYP 4.4?±?3.1 v NOR 2.1?±?2.5?ng.mL^?1 and HYP 117.7?±?86.9 v NOR 72.9?±?85.3?min.ng.mL^?1) in response to HYP. These results suggest that performing high-intensity resistance exercise in a hypoxic environment may provide a beneficial endocrine response without compromising the neuromuscular activation required for maximal strength development.     

A prospective examination of the impact of high levels of exercise training on sedentary behaviour

The aim of this study is to determine changes in sedentary behaviour in response to extensive aerobic exercise training. Participants included adults who self-selected to run a marathon. Sedentary behaviour, total activity counts and physical activity (PA) intensity were assessed (Actigraph GT3X) for seven consecutive days during seven assessment periods (?3, ?2, and ?1 month prior to the marathon, within 2 weeks of the marathon, and +1, +2, and +3 months after the marathon). Models were fitted with multiple imputation data using the STATA mi module. Random intercept generalized least squares (GLS) regression models were used to determine change in sedentary behaviour with seven waves of repeated measures. Results: Twenty-three individuals (mean?±?S_x: 34.4?±?2.1y, 23.0?±?1.9% fat, 15 women, 8 men) completed the study. Marathon finishing times ranged from 185 to 344 minutes (253.2?±?9.6 minutes). Total counts in the vertical axis were 1,729,414 lower one month after the race, compared with two months prior to the race (peak training). Furthermore, counts per minute decreased by 252.7 counts·minute^?1 during that same time period. Daily sedentary behaviour did not change over the seven assessment periods, after accounting for age, gender, per cent body fat, wear time, marathon finishing time, and previous marathon experience. This prospective study supports the notion that PA and sedentary behaviours are distinct, showing that sedentary behaviour was not impacted by high levels of aerobic training.     

Understanding mental toughness in Australian soccer: Perceptions of players,parents, and coaches

Abstract

This study was designed to investigate the effect of ingesting a glucose plus fructose solution on the metabolic responses to soccer-specific exercise in the heat and the impact on subsequent exercise capacity. Eleven male soccer players performed a 90 min soccer-specific protocol on three occasions. Either 3 ml · kg^?1 body mass of a solution containing glucose (1 g · min^?1 glucose) (GLU), or glucose (0.66 g · min^?1) plus fructose (0.33 g · min^?1) (MIX) or placebo (PLA) was consumed every 15 minutes. Respiratory measures were undertaken at 15-min intervals, blood samples were drawn at rest, half-time and on completion of the protocol, and muscle glycogen concentration was assessed pre- and post-exercise. Following the soccer-specific protocol the Cunningham and Faulkner test was performed. No significant differences in post-exercise muscle glycogen concentration (PLA, 62.99 ± 8.39 mmol · kg wet weight^?1; GLU 68.62 ± 2.70; mmol · kg wet weight^?1 and MIX 76.63 ± 6.92 mmol · kg wet weight^?1) or exercise capacity (PLA, 73.62 ± 8.61 s; GLU, 77.11 ± 7.17 s; MIX, 83.04 ± 9.65 s) were observed between treatments (P > 0.05). However, total carbohydrate oxidation was significantly increased during MIX compared with PLA (P < 0.05). These results suggest that when ingested in moderate amounts, the type of carbohydrate does not influence metabolism during soccer-specific intermittent exercise or affect performance capacity after exercise in the heat.     

The effects of an increased calorie breakfast consumed prior to simulated match-play in Academy soccer players

Dietary analysis of Academy soccer players highlights that total energy and carbohydrate intakes are less than optimal, especially, on match-days. As UK Academy matches predominantly kick-off at ～11:00?h, breakfast is likely the last pre-exercise meal and thus may provide an intervention opportunity on match-day. Accordingly, the physiological and performance effects of an increased calorie breakfast consumed ～135-min before soccer-specific exercise was investigated. English Premier League Academy soccer players (n?=?7) repeated a 90-min soccer match simulation on two occasions after consumption of habitual (B_hab; ～1100?kJ) or increased (B_inc; ～2100?kJ) energy breakfasts standardised for macronutrient contributions (～60% carbohydrates, ～15% proteins and ～25% fats). Countermovement jump height, sprint velocities (15-m and 30-m), 30-m repeated sprint maintenance, gut fullness, abdominal discomfort and soccer dribbling performances were measured. Blood samples were taken at rest, pre-exercise, half-time and every 15-min during exercise. Although dribbling precision (P?=?.522; 29.9?±?5.5?cm) and success (P?=?.505; 94?±?8%) were unchanged throughout all time-points, mean dribbling speed was faster (4.3?±?5.7%) in B_inc relative to B_hab (P?=?.023; 2.84 vs 2.75?m?s^?1). Greater feelings of gut fullness (67?±?17%, P?=?.001) were observed in B_inc without changes in abdominal discomfort (P?=?.595). All other physical performance measures and blood lactate and glucose concentrations were comparable between trials (all P?>?.05). Findings demonstrate that Academy soccer players were able to increase pre-match energy intake without experiencing abdominal discomfort; thus, likely contributing to the amelioration of energy deficits on match-days. Furthermore, whilst B_inc produced limited benefits to physical performance, increased dribbling speed was identified, which may be of benefit to match-play.     

Specificity of a Maximal Step Exercise Test

To adhere to the principle of “exercise specificity” exercise testing should be completed using the same physical activity that is performed during exercise training. The present study was designed to assess whether aerobic step exercisers have a greater maximal oxygen consumption (max VO₂) when tested using an activity specific, maximal step exercise test (SET; arms and legs) versus a maximal running test (legs only). Female aerobic step exercisers (N=18; 20.7 ± 1.5 years) performed three maximal graded exercise tests (GXTs): 2 SETs; 1 treadmill test (TMT). The SET consisted of six 3-min progressive stages of alternate lead, basic step, basic step with biceps curls, knee raise with pull-down, repeater knee with pull-down, lateral lunge with pull-down, and side squat with shoulder presses. Stepping rate was 32 steps· min^?1 on an 8-in (20.32 cm) step for stages 1–3, and a 10-in (25.4 cm) step for stages 4–6. Submaximal and maximal heart rate (HR) and oxygen consumption (VO₂) were recorded at the end of each stage. Test–retest reliability for the first five stages of the SET ranged from .91 to .97 for HR, and from .84 to .96 for VO₂. Maximal HR was significantly greater (p =.0001) for the SET (200 ± 6.2 beats·min^?1) as compared to the TMT (193 ± 7.9 beats·min^?1). No significant difference was found for max VO₂ (42.9 ± 8.5, 41.2 ± 5.9 ml·kg^?1·min^?1, p =.14). The SET was a valid and reliable protocol for assessing responses of these aerobic step exercisers; however, max VO₂ from a TMT did not differ significantly from the SET. Conversely, max HR obtained from the criterion TMT was 7 beats·min^?1 lower than from the SET. If a training HR for step exercise (arms and legs exercise) is prescribed based on the max HR from treadmill exercise (legs only), then the training HR should be calculated from a TMT max HR that has been increased by 7 beats·min^?1 to obtain an intensity of step exercise comparable to that of running.     

The influence of upper-body pre-cooling on repeated sprint performance in moderate ambient temperatures

In this study, we examined the effects of upper-body pre-cooling before intermittent sprinting exercise in a moderate environment. Seven male and three female trained cyclists (age 26.8±5.5 years, body mass 68.5±9.5?kg, height 1.76±0.13?m, [Vdot]O_2peak 59.0±11.4?mL?·?kg^?1?·?min^?1; mean±s) performed 30?min of cycling at 50% [Vdot]O_2peak interspersed with a 10-s Wingate cycling sprint test at 5?min intervals. The exercise was performed in a room controlled at 22^oC and 40% relative humidity. In the control session, the participants rested for 30?min before exercise. In the pre-cooling session, the participants wore the upper segment of a liquid conditioning garment circulating 5^oC coolant until rectal temperature decreased by 0.5^oC. Rectal temperature at the start of exercise was significantly lower in the pre-cooling (36.5±0.3^oC) than in the control condition (37.0±0.5^oC), but this difference was reduced to a non-significant 0.4^oC throughout exercise. Mean skin temperature was significantly lower in the pre-cooling (30.7±2.3^oC) than in the control condition (32.5±1.6^oC) throughout exercise. Heart rate during submaximal exercise was similar between the two conditions, although peak heart rate after the Wingate sprints was significantly lower in the pre-cooling condition. With pre-cooling, mean peak power (909±161?W) and mean overall power output (797±154?W) were similar to those in the control condition (peak 921±163?W, mean 806±156?W), with no differences in the subjective ratings of perceived exertion. These results suggest that upper-body pre-cooling does not provide any benefit to intermittent sprinting exercise in a moderate environment.     

Protection against muscle damage in competitive sports players: the effect of the immunomodulator AM3

Strenuous physical exercise of the limb muscles commonly results in damage, especially when that exercise is intense, prolonged and includes eccentric contractions. Many factors contribute to exercise-induced muscle injury and the mechanism is likely to differ with the type of exercise. Competitive sports players are highly susceptible to this type of injury. AM3 is an orally administered immunomodulator that reduces the synthesis of proinflammatory cytokines and normalizes defective cellular immune fractions. The ability of AM3 to prevent chronic muscle injury following strenuous exercise characterized by eccentric muscle contraction was evaluated in a double-blind and randomized pilot study. Fourteen professional male volleyball players from the First Division of the Spanish Volleyball League volunteered to take part. The participants were randomized to receive either placebo (n?=?7) or AM3 (n?=?7). The physical characteristics (mean±s) of the placebo group were as follows: age 25.7±2.1 years, body mass 87.2±4.1?kg, height 1.89±0.07?m, maximal oxygen uptake 65.3±4.2?ml?·?kg^?1?·?min^?1. Those of the AM3 group were as follows: age 26.1±1.9 years, body mass 85.8±6.1?kg, height 1.91±0.07?m, maximal oxygen uptake 64.6±4.5?ml?·?kg^?1?·?min^?1. All participants were evaluated for biochemical indices of muscle damage, including concentrations of aspartate aminotransferase, alanine aminotransferase, alkaline phosphatase, creatine kinase (CK) and its MB fraction (CK-MB), myoglobin, lactate dehydrogenase, urea, creatinine and γ-glutamyltranspeptidase, both before and 30 days after treatment (over the peak of the competitive season). In the placebo group, competitive exercise (i.e. volleyball) was accompanied by significant increases in creatine kinase (494±51 to 560±53?IU?·?l^?1, P?<?0.05) and myoglobin (76.8±2.9 to 83.9±3.1?μg?·?l^?1, P?<?0.05); aspartate aminotransferase (30.8±3.0 to 31.1±2.9?IU?·?l^?1) and lactate dehydrogenase (380±31 to 376±29?IU?·?l^?1) were relatively unchanged after the 30 days maximum effort. AM3 not only inhibited these changes, it led to a decrease from baseline serum concentrations of creatine kinase (503±49 to 316±37?IU?·?l^?1, P?<?0.05) and myoglobin (80.1±3.2 to 44.1±2.6?IU?·?l^?1, P?<?0.05), as well as aspartate aminotransferase (31.1±3.3 to 26.1±2.7?IU?·?l^?1, P?<?0.05) and lactate dehydrogenase (368±34 to 310±3?IU?·?l^?1, P?<?0.05). The concentration of CK-MB was also significantly decreased from baseline with AM3 treatment (11.6±1.2 to 5.0±0.7?IU?·?l^?1, P?<?0.05), but not with placebo (11.4±1.1 to 10.8±1.4?IU?·?l^?1). In conclusion, the use of immunomodulators, such as AM3, by elite sportspersons during competition significantly reduces serum concentrations of proteins associated with muscle damage.     

Larger strength losses and muscle activation deficits in plantar flexors induced by backward downhill in reference to distance-matched forward uphill treadmill walk

We tested the hypothesis that backward downhill walking (eccentric component) impairs both voluntary activation and muscle contractile properties in the plantar flexors and delays recovery as compared to a gradient and distance-matched uphill walk. Fourteen males performed two 30-min walking exercises (velocity: 1?m/?s; grade: 25%; load: 12% of body weight), one downhill (DW) and one uphill (UP), in a counterbalanced order, separated by 6?weeks. Neuromuscular test sessions were performed before, after, 24-, 48- and 72-h post-exercise, including motor nerve stimulations during brief (5?s) and sustained (1?min) maximal isometric voluntary contractions of the plantar flexors. DW (?18.1?±?11.1%, P?P?=.15), decreased torque production during brief contractions for at least three days post-exercise (P?P?P?=?.024) and DW (?25.6?±?10.3%, P?P?=?.001) was lower in DW than UP. Peak twitch torque and maximum rates of torque development and relaxation were equally reduced after UP and DW (P?P?P?>?.05). Using a direct comparison, the capacity to drive the plantar flexors during sustained contractions remains sub-optimal during the three-day recovery period in response to non-exhaustive, downhill backward walking in reference to an uphill exercise matched for distance covered.     

Effect of repeat-sprint training in hypoxia on post-exercise interleukin-6 and F2-isoprostanes

This investigation examined the oxidative stress (F₂-Isoprostane; F₂-IsoP) and inflammatory (interleukin-6; IL-6) responses to repeat-sprint training in hypoxia (RSH). Ten trained male team sport athletes performed 3(sets)*9(repetitions)*5?s cycling sprints in simulated altitude (3000?m) and sea-level conditions. Mean and peak sprint power output (MPO and PPO) were recorded, and blood samples were collected pre-exercise, and again at 8 and 60?min post-exercise. Both MPO and PPO were significantly reduced in hypoxia (compared to sea-level) in the second (MPO: 855?±?89 vs. 739?±?95?W, p?=?.006; PPO: 1024?±?114 vs. 895?±?112?W, p?=?.010) and third (MPO: 819?±?105 vs. 686?±?83?W, p?=?.008; PPO: 985?±?125 vs. 834?±?99?W, p?=?.008) sets, respectively. IL-6 was significantly increased from pre- to 1?h post-exercise in both hypoxia (0.7?±?0.2 vs. 2.4?±?1.4?pg/mL, p?=?.004) and sea-level conditions (0.7?±?0.2 vs. 1.6?±?0.3?pg/mL, p?d?=?0.80) suggesting higher IL-6 levels of post-hypoxia. F₂-IsoP was significantly lower 1?h post-exercise in both the hypoxic (p?=?.005) and sea-level (p?=?.002) conditions, with no differences between trials. While hypoxia can impact on exercise intensity and may result in greater post-exercise inflammation, it appears to have little effect on oxidative stress. These results indicate that team sport organisations with ready access to hypoxic training facilities could confidently administer RSH without significantly increasing the post-exercise inflammatory or oxidative stress response.     

The relationship between cardiac autonomic function and maximal oxygen uptake response to high-intensity intermittent-exercise training

Abstract

Major individual differences in the maximal oxygen uptake response to aerobic training have been documented. Vagal influence on the heart has been shown to contribute to changes in aerobic fitness. Whether vagal influence on the heart also predicts maximal oxygen uptake response to interval-sprinting training, however, is undetermined. Thus, the relationship between baseline vagal activity and the maximal oxygen uptake response to interval-sprinting training was examined. Exercisers (n?=?16) exercised three times a week for 12 weeks, whereas controls did no exercise (n?=?16). Interval-sprinting consisted of 20 min of intermittent sprinting on a cycle ergometer (8 s sprint, 12 s recovery). Maximal oxygen uptake was assessed using open-circuit spirometry. Vagal influence was assessed through frequency analysis of heart rate variability. Participants were aged 22 ± 4.5 years and had a body mass of 72.7 ± 18.9 kg, a body mass index of 26.9 ± 3.9 kg · m^?2, and a maximal oxygen uptake of 28 ± 7.4 ml · kg^?1 · min^?1. Overall increase in maximal oxygen uptake after the training programme, despite being anaerobic in nature, was 19 ± 1.2%. Change in maximal oxygen uptake was correlated with initial baseline heart rate variability high-frequency power in normalised units (r?=?0.58; P < 0.05). Thus, cardiac vagal modulation of heart rate was associated with the aerobic training response after 12 weeks of high-intensity intermittent-exercise. The mechanisms underlying the relationship between the aerobic training response and resting heart rate variability need to be established before practical implications can be identified.     

Muscle fatigue induced by exercise simulating the work rate of competitive soccer

Fatigue represents a reduction in the capability of muscle to generate force. The aim of the present study was to establish the effects of exercise that simulates the work rate of competitive soccer players on the strength of the knee extensors and knee flexors. Thirteen amateur soccer players (age 23.3±3.9 years, height 1.78±0.05?m, body mass 74.8±3.6?kg; mean±s) were tested during the 2000–2001 soccer season. Muscle strength of the quadriceps and hamstrings was measured on an isokinetic dynamometer. A 90?min soccer-specific intermittent exercise protocol, incorporating a 15?min half-time intermission, was developed to provide fatiguing exercise corresponding in work rate to a game of soccer. The exercise protocol, performed on a programmable motorized treadmill, consisted of the different intensities observed during soccer match-play (e.g. walking, jogging, running, sprinting). Muscle strength was assessed before exercise, at half-time and immediately after exercise. A repeated-measures analysis of variance showed significant reductions (P?<0.001) in peak torque for both the quadriceps and hamstrings at all angular velocities (concentric: 1.05, 2.09, 5.23 rad?·?s^?1; eccentric: 2.09 rad?·?s^?1). The peak torque of the knee extensors (KE) and knee flexors (KF) was greater before exercise [KE: 232±37, 182±34, 129±27, 219±41?N?·?m at 1.05, 2.09 and 5.23 rad?·?s^?1 (concentric) and 2.09 rad?·?s^?1 (eccentric), respectively; KF: 126±20, 112±19, 101±16, 137±23?N?·?m] than at half-time (KE: 209±45, 177±35, 125±36, 214±43?N?·?m; KF: 114±31, 102±20, 92±15, 125±25?N?·?m) and greater at half-time than after exercise (KE: 196±43, 167±35, 118±24, 204±43?N?·?m; KF: 104±25, 95±21, 87±13, 114±27?N?·?m). For the hamstrings?:?quadriceps ratio, significant changes were found (P?<0.05) for both legs, the ratio being greater before than after exercise. For fast?:?slow speed and left?:?right ratios, no significant changes were found. We conclude that there is a progressive reduction in muscle strength that applies across a range of functional characteristics during exercise that mimics the work rate in soccer.     

Sweat lactate response during cycling at 30°C and 18°C WBGT

Sweat lactate reflects eccrine gland metabolism. However, the metabolic tendencies of eccrine glands in a hot versus thermoneutral environment are not well understood. Sixteen male volunteers completed a maximal cycling trial and two 60-min cycling trials [30°C?=?30±1°C and 18°C?=?18±1°C wet bulb globe temperature (WBGT)]. The participants were requested to maintain a cadence of 60 rev?·?min^?1 with the intensity individualized at ～ 90% of the ventilatory threshold. Sweat samples at 10, 20, 30, 40, 50 and 60?min were analysed for lactate concentration. Sweat rate at 30°C (1380±325?ml?·?h^?1) was significantly greater (P<0.05) than at 18°C (632±311?ml?·?h^?1). Sweat lactate concentration was significantly greater (P<0.05) at each time point during the 18°C trial, with values between trials tending to converge across time. During the 30°C trial, both heart rate (20, 30, 40, 50 and 60?min) and rectal temperature (30, 40, 50 and 60?min) were significantly higher than in the 18°C trial. Higher sweat lactate concentrations coupled with lower sweat rates may indicate a greater relative contribution of oxygen-independent metabolism within eccrine glands during exercise at 18°C. Decreases in sweat lactate concentration across time suggest either greater dilution due to greater sweat volume or increased reliance on aerobic metabolism within eccrine glands. The convergence of lactate concentrations between trials may indicate that time-dependent modifications in sweat gland metabolism occur at different rates contingent partially on environmental conditions.     

Effects of whey protein in carbohydrate-electrolyte drinks on post-exercise rehydration

The purpose of this study was to examine the effects of different amounts of whey protein in carbohydrate–electrolyte (CE) drinks on post-exercise rehydration. Ten males completed 5 trials in a randomised cross-over design. A 4-h recovery was applied after a 60-min run at 65% VO_2peak in each trial. During recovery, the participants ingested a high-carbohydrate CE drink (CE-H), a low-carbohydrate CE drink (CE-L), a high-whey-protein (33?g·L^?1) CE drink (CW-H), a medium-whey-protein (22?g·L^?1) CE drink (CW-M) or a low-whey-protein (15?g·L^?1) CE drink (CW-L) in a volume equivalent to 150% of their body mass (BM) loss. The drinks were provided in six equal boluses and consumed by the participants within 150?min in each trial. After exercise, a BM loss of 2.15%?±?0.05% was achieved. Urine production was less in the CW-M and CW-H trials during recovery, which induced a greater fluid retention in the CW-M (51.0%?±?5.7%) and CW-H (55.4%?±?3.8%) trials than in any other trial (p?p?p?p??1 in the current study.