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运动、个体性特征与心脏自主功能 总被引:1,自引:0,他引:1
巩绪伟 《山东体育学院学报》2010,26(6)
耐力训练能够降低静息状态下和次强度运动下的心率,但是最大心率只是轻微下降或保持不变.近来的研究结果支持这一假说:评定自主神经系统的功能能够获得有氧训练刺激前、中和后关于急性或长期生理变化过程重要的信息;日常评定自主神经系统的功能活性能够为个体有氧训练适宜的生理条件提供指示. 相似文献
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运动作为一种很好的心脏保护方式,对心血管系统的潜在效应是复杂而矛盾的.自主神经功能调节与心血管疾病密切相关.提高迷走张力,维持自主神经平衡对于心血管疾病的治疗、康复及预防都至关重要.但运动对自主神经的心肌保护作用及机制是什么,不同运动强度、方式对心肌自主神经功能调节的影响规律还不清楚. 相似文献
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《运动与健康科学(英文)》2020,9(5):432-445
Several decades of research in the area of exercise immunology have shown that the immune system is highly responsive to acute and chronic exercise training. Moderate exercise bouts enhance immunosurveillance and when repeated over time mediate multiple health benefits. Most of the studies prior to 2010 relied on a few targeted outcomes related to immune function. During the past decade, technologic advances have created opportunities for a multi-omics and systems biology approach to exercise immunology. This article provides an overview of metabolomics, lipidomics, and proteomics as they pertain to exercise immunology, with a focus on immunometabolism. This review also summarizes how the composition and diversity of the gut microbiota can be influenced by exercise, with applications to human health and immunity. Exercise-induced improvements in immune function may play a critical role in countering immunosenescence and the development of chronic diseases, and emerging omics technologies will more clearly define the underlying mechanisms. This review summarizes what is currently known regarding a multi-omics approach to exercise immunology and provides future directions for investigators. 相似文献
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本文综述了当前运动对Lepfin效应的主要研究工作。Lepfin是近年发现的能量平衡调节的激素,是Ob基因的表达产物。运动对Leptin水平的影响表现在:长期有氧运动可降低血Lepfin浓度,减少脂肪组织Ob基因的表达,而一次性运动对Lepdn表达和其血中浓度的影响研究结果不一。运动的这些效应可能依赖于运动负荷量和运动时间。 相似文献
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Exercise in the heat poses a formidable challenge to the body's ability to control its internal environment due to the high rates of metabolic heat production and heat gain by physical transfer from the environment. In an attempt to restrict the rise in core temperature, an increased rate of sweat secretion onto the skin is invoked. This may limit the rise in core temperature, and can prolong the time before a limiting temperature is attained, but it does so at the cost of a loss of body water and electrolytes. The effects of the diminished blood volume are offset to some extent by cardiovascular adaptations, including an increased heart rate and an increased peripheral resistance, but these are insufficient to maintain functional capacity when blood volume is reduced. Prior dehydration will impair performance in both prolonged exercise and short-term high-intensity exercise. Athletes living and training in the heat may experience chronic hypohydration due to inadequate replacement of fluid losses. The negative consequences of exercise in the heat are attenuated to some extent by a period of adaptation, and by the ingestion of water or other appropriate fluids. Optimum fluid replacement strategies will depend on the exercise task, the environmental conditions and the individual physiological characteristics of the athlete. Manipulation of pre-exercise body temperature can also influence exercise performance and may be a strategy that can be used by athletes competing in stressful environments. 相似文献
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Exercise in the heat poses a formidable challenge to the body's ability to control its internal environment due to the high rates of metabolic heat production and heat gain by physical transfer from the environment. In an attempt to restrict the rise in core temperature, an increased rate of sweat secretion onto the skin is invoked. This may limit the rise in core temperature, and can prolong the time before a limiting temperature is attained, but it does so at the cost of a loss of body water and electrolytes. The effects of the diminished blood volume are offset to some extent by cardiovascular adaptations, including an increased heart rate and an increased peripheral resistance, but these are insufficient to maintain functional capacity when blood volume is reduced. Prior dehydration will impair performance in both prolonged exercise and short-term high-intensity exercise. Athletes living and training in the heat may experience chronic hypohydration due to inadequate replacement of fluid losses. The negative consequences of exercise in the heat are attenuated to some extent by a period of adaptation, and by the ingestion of water or other appropriate fluids. Optimum fluid replacement strategies will depend on the exercise task, the environmental conditions and the individual physiological characteristics of the athlete. Manipulation of pre-exercise body temperature can also influence exercise performance and may be a strategy that can be used by athletes competing in stressful environments. 相似文献
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不同的运动强度红细胞免疫功能有不同的变化;热环境影响红细胞的免疫功能;进行营养素补充对改善红细胞免疫功能有一定的影响. 相似文献
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综述了有关运动与糖皮质激素的研究进展,1.运动与高原训练可引起血中糖皮质激素水平显著升高,并与运动强度有很大关系,而与运动总量和持续时间相关性较小;2.过量糖皮质激素可导致肌肉萎缩,对其发生的机制有新的认识,而运动能否有效预防和治疗该性质的肌肉萎缩,尚有争议;3.有关运动引起糖皮演激素分泌水平改变对机体免疫的影响及其机制。 相似文献
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瘦素与运动 总被引:2,自引:0,他引:2
潘志军 《西安体育学院学报》2003,20(6):29-32
瘦素是由脂肪组织分泌的具有增加能量消耗、减少食物摄取、减少体脂、控制体重和调节能量平衡的一种激素,与体脂含量密切相关,其分泌受多种因素影响。急性运动对血中瘦素的影响有不变和下降的不同结果;长期运动训练可降低血瘦素的含量,这是由于体脂量的下降所致。运动对血瘦素的效应存在着延时性。 相似文献
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瘦素与运动 总被引:1,自引:0,他引:1
魏源 《沈阳体育学院学报》2004,23(6):750-752
瘦素(leptin)是主要由脂肪组织分泌的一种激素,由146个氨基酸组成,其前体为167个氨基酸的蛋白质。N-端21肽是信号肽,瘦素作为脂肪组织和中枢神经系统间网络联系的外周信号,可能通过血脑屏障作用于中枢神经系统,影响摄食行为和调整自主神经系统活动等,参与保持机体脂肪量恒定的自稳态调节机制。近年来,国内外针对瘦素的研究涉及各个领域,笔者主要对运动与瘦素变化的关系及其机制进行简要综述。 相似文献
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维生素和矿物质对于维持机体的免疫功能起着重要作用.尤其是微量营养素对免疫功能的影响较大,运动训练使部分运动员可能处于微量营养素缺乏的危险边缘.微量营养素需求量缺乏和过量的后果对机体的免疫功能均有影响,并对提高免疫功能可能存在的机制进行了阐述. 相似文献