Effect of acute ingestion of β-hydroxybutyrate salts on the response to graded exercise in trained cyclists

Acute ingestion of ketone salts induces nutritional ketosis by elevating β-hydroxybutyrate (βHB), but few studies have examined the metabolic effects of ingestion prior to exercise. Nineteen trained cyclists (12 male, 7 female) undertook graded exercise (8 min each at ～30%, 40%, 50%, 60%, 70%, and 80% VO_2peak) on a cycle ergometer on two occasions separated by either 7 or 14 days. Trials included ingestion of boluses of either (i) plain water (3.8?mL?kg?body mass^?1) (CON) or (ii) βHB salts (0.38?g?kg?body mass^?1) in plain water (3.8?mL?kg body mass^?1) (KET), at both 60 min and 15 min prior to exercise. During KET, plasma [βHB] increased to 0.33?±?0.16?mM prior to exercise and 0.44?±?0.15?mM at the end of exercise (both p?.05). Plasma glucose was 0.44?±?0.27?mM lower (p?.01) 30?min after ingestion of KET and remained ～0.2?mM lower throughout exercise compared to CON (p?.001). Respiratory exchange ratio (RER) was higher during KET compared to CON (p?.001) and 0.03–0.04 higher from 30%VO_2peak to 60%VO_2peak (all p?.05). No differences in plasma lactate, rate of perceived exertion, or gross or delta efficiency were observed between trials. Gastrointestinal symptoms were reported in 13 out of 19 participants during KET. Acute ingestion of βHB salts induces nutritional ketosis and alters the metabolic response to exercise in trained cyclists. Elevated RER during KET may be indicative of increased ketone body oxidation during exercise, but at the plasma βHB concentrations achieved, ingestion of βHB salts does not affect lactate appearance, perceived exertion, or muscular efficiency.     

Isolated Obesity Is Not Enough to Impair Cardiac Autonomic Modulation in Metabolically Healthy Men

Purpose: To evaluate whether excess body mass influences the heart rate variability (HRV) indexes at rest, and to correlate adiposity indicators and the aerobic fitness with cardiac autonomic variables in metabolically healthy young adults. Method: In all, 41 untrained males (M_age = 21.80, SD = 2.14 years), 14 normal weight (M_BMI = 22.28, SD = 1.86 kg?m^?2), 11 overweight (M_BMI = 26.95, SD = 1.43 kg?m^?2), and 16 obese (M_BMI = 33.58, SD = 3.06 kg?m^?2) metabolically healthy (normal values of blood pressure, fasting blood glucose, triglycerides, and total cholesterol), underwent evaluations of the HRV at rest and of the peak oxygen consumption (VO₂ peak) during maximal exercise on a cycle ergometer. Results: Blood pressure, heart rate, HRV indexes, casual blood glucose, oxidative stress, and antioxidant activity did not differ among the groups. The VO₂ peak (mL?kg^?1?min^?1) was lower in the obese group compared with the normal weight and overweight groups. The body mass (r = ?.40 to ?.45) and abdominal circumference (r = ?.39 to ?.52) were slightly to moderately correlated with SD1, SD2, RMSSD, SDNN, pNN50, LF, and HF indexes and total power. The VO₂ peak (mL?kg^?1?min^?1) was slightly to moderately correlated (r = .48 to .51) with SD2, SDNN, and LF indexes in the individuals with excess body mass. Conclusion: Cardiac autonomic modulation at rest was preserved in metabolically healthy obese young men. However, the indicators of adiposity, as well as the aerobic fitness were correlated with cardiac autonomic modulation in the individuals with excess body mass.     

Estimating changes in hydration status from changes in body mass: Considerations regarding metabolic water and glycogen storage

Abstract

The potential for imprecision in the estimation of hydration status from changes in body mass has been outlined previously but the equations derived from these derivations appear inconsistent. Reconciliation of body mass loss in terms of sweat loss and effective body water loss is possible from specific equation sets provided that gains and losses of both body mass and water used in the derivation of sweat loss and to derive effective body water loss are in inclusive equation sets. This is obligatory so that mass and water changes as quantifiable determinants are consistent with both internal processes and external gains and losses. Thus, body mass loss, substrate oxidation, metabolic water, and all the terms used in simultaneous equation sets have to be reconciled not only as identical variables but mathematically balance exactly. The revised equation for effective body water loss given here is different from that originally proposed. Metabolic water is part of body mass loss corrected for substrate oxidation, fluid ingestion, and respiratory water to derive sweat loss and it may not be justified to also include water associated with glycogen as releasable bound water. Accordingly, our calculated effective body water loss is substantially a greater loss than originally supposed but clearly still less than the simple balance between mass loss and fluid ingested.     

Tear osmolarity is sensitive to exercise-induced fluid loss but is not associated with common hydration measures in a field setting

This investigation (i) examined changes in tear osmolarity in response to fluid loss that occurs with exercise in a field setting, and (ii) compared tear osmolarity with common field and laboratory hydration measures. Sixty-three participants [age 27.8 ± 8.4 years, body mass 72.15 ± 10.61 kg] completed a self-paced 10 km run outside on a predetermined course. Body mass, tear fluid, venous blood and urine samples were collected immediately before and after exercise. Significant (p < 0.001) reductions in body mass (1.71 ± 0.44%) and increases in tear osmolarity (8 ± 15 mOsm.L^?1), plasma osmolality (7 ± 8 mOsm.kg^?1), and urine specific gravity (0.0014 ± 0.0042 g.mL^?1; p = 0.008) were observed following exercise. Pre- to post-exercise change in tear osmolarity was not significantly correlated (all p > 0.05) with plasma osmolality (r_s = 0.24), urine osmolality (r_s = 0.14), urine specific gravity (r_s = 0.13) or relative body mass loss (r = 0.20). Tear osmolarity is responsive to exercise-induced fluid loss but does not correlate with the changes observed using other common measures of hydration status in the field setting. Practitioners shouldn’t directly compare or replace other common hydration measures with tear osmolarity in the field.

Abbreviations: BML: Body Mass Loss; CV: Coefficient of Variation; P_osm: Plasma osmolality; SD: Standard Deviation; T_osm: Tear Osmolarity; U_osm: Urine Osmolality; USG: Urine Specific Gravity; WBGT: Wet bulb globe thermometer     

Ergogenic effects of precooling with cold water immersion and ice ingestion: A meta-analysis

This review evaluated the effects of precooling via cold water immersion (CWI) and ingestion of ice slurry/slushy or crushed ice (ICE) on endurance performance measures (e.g. time-to-exhaustion and time trials) and psychophysiological parameters (core [T_core] and skin [T_skin] temperatures, whole body sweat [WBS] response, heart rate [HR], thermal sensation [TS], and perceived exertion [RPE]). Twenty-two studies were included in the meta-analysis based on the following criteria: (i) cooling was performed before exercise with ICE or CWI; (ii) exercise longer than 6?min was performed in ambient temperature ≥26°C; and (iii) crossover study design with a non-cooling passive control condition. CWI improved performance measures (weighted average effect size in Hedges’ g [95% confidence interval]?+?0.53 [0.28; 0.77]) and resulted in greater increase (ΔEX) in T_skin (+4.15 [3.1; 5.21]) during exercise, while lower peak T_core (?0.93 [?1.18; ?0.67]), WBS (?0.74 [?1.18; ?0.3]), and TS (?0.5 [?0.8; ?0.19]) were observed without concomitant changes in ΔEX-T_core (+0.19 [?0.22; 0.6]), peak T_skin (?0.67 [?1.52; 0.18]), peak HR (?0.14 [?0.38; 0.11]), and RPE (?0.14 [?0.39; 0.12]). ICE had no clear effect on performance measures (+0.2 [?0.07; 0.46]) but resulted in greater ΔEX-T_core (+1.02 [0.59; 1.45]) and ΔEX-T_skin (+0.34 [0.02; 0.67]) without concomitant changes in peak T_core (?0.1 [?0.48; 0.28]), peak T_skin (+0.1 [?0.22; 0.41]), peak HR (+0.08 [?0.19; 0.35]), WBS (?0.12 [?0.42; 0.18]), TS (?0.2 [?0.49; 0.1]), and RPE (?0.01 [?0.33; 0.31]). From both ergogenic and thermoregulatory perspectives, CWI may be more effective than ICE as a precooling treatment prior to exercise in the heat.     

Seasonal changes in body composition of inter-county Gaelic Athletic Association hurlers

Longitudinal change in body composition for elite-level inter-county hurlers was reported over a single season and four consecutive seasons. Body composition measured by dual-energy x-ray absorptiometry (DXA) of 66 senior, male, outfield players was obtained. Four successive measurements were taken: off-season (OFF₁), pre-season (PRE), mid-season (MID) and the off-season of the following season (OFF₂). A subsample of 11 hurlers were measured at all time points over 4 consecutive seasons. DXA-derived estimates of fat and lean mass were normalised to stature for analysis (kg?m^?2); data are (mean [lower: upper, 95% confidence interval]). A concurrent increase of lean mass (0.31 [0.19: 0.43] kg?m^?2) and loss of fat mass occurred (?0.38 [?0.50: ?0.26] kg?m^?2) OFF₁ to PRE. Lean mass accrual was maintained PRE to OFF₂ while the initial loss of fat mass was restored MID to OFF₂ (0.52 [0.40: 0.64] kg ? m^?2), with the trunk acting as the primary region of change. Over the four seasons, a net increase of lean mass was observed (~ 0.9 [0.4: 1.4] kg per annum) with a negligible overall change for fat mass over time. However, the cycling of fat mass (OFF to PRE and MID to OFF) within each season was recurrent season-to-season.     

A faster running speed is associated with a greater body weight loss in 100-km ultra-marathoners

Abstract

In 219 recreational male runners, we investigated changes in body mass, total body water, haematocrit, plasma sodium concentration ([Na⁺]), and urine specific gravity as well as fluid intake during a 100-km ultra-marathon. The athletes lost 1.9 kg (s = 1.4) of body mass, equal to 2.5% (s = 1.8) of body mass (P < 0.001), 0.7 kg (s = 1.0) of predicted skeletal muscle mass (P < 0.001), 0.2 kg (s = 1.3) of predicted fat mass (P < 0.05), and 0.9 L (s = 1.6) of predicted total body water (P < 0.001). Haematocrit decreased (P < 0.001), urine specific gravity (P < 0.001), plasma volume (P < 0.05), and plasma [Na⁺] (P < 0.05) all increased. Change in body mass was related to running speed (r = ?0.16, P < 0.05), change in plasma volume was associated with change in plasma [Na⁺] (r = ?0.28, P < 0.0001), and change in body mass was related to both change in plasma [Na⁺] (r = ?0.36) and change in plasma volume (r = 0.31) (P < 0.0001). The athletes consumed 0.65 L (s = 0.27) fluid per hour. Fluid intake was related to both running speed (r = 0.42, P < 0.0001) and change in body mass (r = 0.23, P = 0.0006), but not post-race plasma [Na⁺] or change in plasma [Na⁺] (P > 0.05). In conclusion, faster runners lost more body mass, runners lost more body mass when they drank less fluid, and faster runners drank more fluid than slower runners.     

Effect of water ingestion on endurance capacity during prolonged running

This study examined the influence of water ingestion on endurance capacity during submaximal treadmill running. Four men and four women with a mean (± S.E.) age of 21.4 ± 0.7 years, height of 169 + 2 cm, body mass of 63.1 ± 2.9 kg and VO ₂ max of 51.1 ± 1.8 ml kg^?1 min^?1, performed two randomly assigned treadmill runs at 70% VO ₂ max to exhaustion. No fluid was ingested during one trial (NF‐trial), whereas a single water bolus of 3.0 ml kg^?1 body mass was ingested immediately pre‐exercise and serial feedings of 2.0 ml kg^?1 body mass were ingested every 15 min during exercise in a fluid replacement trial (FR‐trial). Run time for the NF‐trial was 77.7 ± 7.7 min, compared to 103 ± 12.4 min for the FR‐trial (P<0.01). Body mass (corrected for water ingestion) decreased by 2.0 ± 0.2% in the NF‐trial and 2.7 ± 0.2% in the FR‐trial (P<0.01), while plasma volume decreased by 1.1 ± 1.1% and 3.5 ± 1.1% in the two trials respectively (N.S.). However, these apparent differences in circulatory volume were not associated with differences in rectal temperature. Respiratory exchange ratios indicated increased carbohydrate metabolism (73% vs 64% of total energy expenditure) and suppressed fat metabolism after 75 min of exercise in the NF‐trial compared with the FR‐trial (NF‐trial, 0.90 ± 0.01; FR‐trial, 0.86 ± 0.03; P<0.01). Blood glucose concentrations were similar in both trials, while blood lactate concentrations were higher in the NF‐trial at the end of exercise (4.83 ± 0.34 vs 4.18 ± 0.38 mM; P<0.05). In summary, water ingestion during prolonged running improved endurance capacity.     

Effect of Mouthguard Use on Metabolic and Cardiorespiratory Responses to Aerobic Exercise in Males

Purpose: This study investigated the physiological effects of wearing a mouthguard during submaximal treadmill exercise. Method: Twenty-four recreationally active males (M_age = 21.3 ± 2.4 years, M_height = 1.78 ± 0.06 m, M_weight = 81.9 ± 10.6 kg, M_{body mass index} = 25.8 ± 3.4 kg·m^?2) performed incremental, continuous exercise at 2, 4, 6, and 8 mph (3.2, 6.4, 9.7, 12.9 kph) for 5 min at each speed on a motor-driven treadmill on 2 separate occasions in a randomized, crossover, counterbalanced design while wearing or not wearing a self-adaptable “boil and bite” mouthguard. Respiratory rate (RR), tidal volume (V_T), ventilation (V_E), oxygen consumption (VO₂), respiratory exchange ratio (RER), and heart rate (HR) data were averaged during the last 60 s of each exercise stage; blood lactate (LA) was measured before exercise and 3 min and 10 min following exercise. Results: Repeated-measures analysis of variance revealed that mouthguard use failed to alter the response of RR, V_T, V_E, VO₂, RER, and HR to treadmill exercise (p > .05), although each variable did increase in magnitude as a result of increasing treadmill speed (p < .001). Although increasing to above resting values at both 3 min and 10 min (p < .001) after cessation of exercise, LA levels also displayed no differences with mouthguard use (p > .05). Conclusion: Despite predictable increases in respiratory, metabolic, and cardiovascular variables in response to incremental exercise, the presence of a mouthguard failed to affect the magnitude or nature of these physiological responses.     

Body composition of female road and track endurance cyclists: Normative values and typical changes

The aims of this study were to describe normative values and seasonal variation of body composition in female cyclists comparing female road and track endurance cyclists, and to validate the use of anthropometry to monitor lean mass changes. Anthropometric profiles (seven site skinfolds) were measured over 16 years from 126 female cyclists. Lean mass index (LMI) was calculated as body weight?×?skinfolds^?x. The exponent (x) was calculated as the slope of the natural logarithm of body weight and skinfolds. Percentage changes in LMI were compared to lean mass changes measured using dual-energy X-ray absorptiometry (DXA) in a subset of 25 road cyclists. Compared to sub-elite and elite cyclists, world class cyclists were (mean [95% CI]) 1.18?kg [0.46, 1.90] and 0.60?kg [0.05, 1.15] lighter and had skinfolds that were 7.4?mm [3.8, 11.0] and 4.6?mm [1.8, 7.4] lower, respectively. Body weight (0.41?kg [0.04, 0.77]) and skinfolds (4.0?mm [2.1, 6.0]) were higher in the off-season compared to the early-season. World class female road cyclists had lower body weight (6.04?kg [2.73, 9.35]) and skinfolds (11.5?mm [1.1, 21.9]) than track endurance cyclists. LMI (mean exponent 0.15 [0.13, 0.18]) explained 87% of the variance in DXA lean mass. In conclusion, higher performing female cyclists were lighter and leaner than their less successful peers, road cyclists were lighter and leaner than track endurance cyclists, and weight and skinfolds were lowest early in the season. LMI appears to be a reasonably valid tool for monitoring lean mass changes.     

Thermoneutral immersion exercise accelerates heart rate recovery: A potential novel training modality

This study compared heart rate recovery (HRR) after incremental maximal exercise performed at the same external power output (P_ext) on dry land ergocycle (DE) vs. immersible ergocycle (IE). Fifteen young healthy participants (30?±?7 years, 13 men and 2 women) performed incremental maximal exercise tests on DE and on IE. The initial P_ext on DE was 25?W and was increased by 25?W/min at a pedalling cadence between 60 and 80?rpm, while during IE immersion at chest level in thermoneutral water (30°C), the initial P_ext deployment was at a cadence of 40?rpm which was increased by 10?rpm until 70?rpm and thereafter by 5?rpm until exhaustion. Gas exchange and heart rate (HR) were measured continuously during exercise and recovery for 5?min. Maximal HR (DE: 176?±?15 vs. IE 169?±?12?bpm) reached by the subjects in the two conditions did not differ (P?>?.05). Parasympathetic reactivation parameters (ΔHR from 10 to 300?s) were compared during the DE and IE HR recovery recordings. During the IE recovery, parasympathetic reactivation in the early phase was more predominant (HRR at Δ10–Δ60?s, P?<?.05), but similar in the late phase (HRR at Δ120–Δ300?s, P?>?.05) when compared to the DE condition. In conclusion, incremental maximal IE exercise at chest level immersion in thermoneutral water accelerates the early phase parasympathetic reactivation compared to DE in healthy young participants.     

急性低氧对优秀男子自行车运动员递增负荷运动时肌氧含量的影响

目的:探讨急性低氧环境下递增负荷运动时肌氧含量的变化。方法:通过无损伤近红外光谱学技术(NIRS)监测11名优秀男子自行车运动员常氧和急性低氧下进行递增负荷运动时肌氧含量和心肺系统功能的变化。结果:1)低氧运动时,无氧阈(VT)和最大摄氧量(VO2max)出现时对应的心率、指动脉血氧饱和度(SpO2)、气体代谢和功率均下降,显著低于常氧运动时的值;2)低氧环境下,从开始运动到75%最大功率,Δ[HbO2]降低、Δ[HHb]增高;由75%至100%最大功率时,Δ[HbO2]保持不变,而Δ[HHb]和Δ[THb]增加;在4个不同功率等级下低氧Δ[HbO2]均显著高于常氧值,Δ[HHb]在50%、75%和100%最大功率时均显著低于常氧对应值。结论:1)男子自行车运动员低氧下运动时低氧通气反应高于常氧水平,说明提高自行车运动员在高原训练时的低氧通气反应有利于提高其有氧能力,进而提高运动成绩。2)低氧运动时Δ[HbO2]高于常氧值,Δ[HHb]低于常氧值,提示肌氧含量是反映肌肉疲劳程度的敏感指标。     

The influence of upper-body pre-cooling on repeated sprint performance in moderate ambient temperatures

In this study, we examined the effects of upper-body pre-cooling before intermittent sprinting exercise in a moderate environment. Seven male and three female trained cyclists (age 26.8±5.5 years, body mass 68.5±9.5?kg, height 1.76±0.13?m, [Vdot]O_2peak 59.0±11.4?mL?·?kg^?1?·?min^?1; mean±s) performed 30?min of cycling at 50% [Vdot]O_2peak interspersed with a 10-s Wingate cycling sprint test at 5?min intervals. The exercise was performed in a room controlled at 22^oC and 40% relative humidity. In the control session, the participants rested for 30?min before exercise. In the pre-cooling session, the participants wore the upper segment of a liquid conditioning garment circulating 5^oC coolant until rectal temperature decreased by 0.5^oC. Rectal temperature at the start of exercise was significantly lower in the pre-cooling (36.5±0.3^oC) than in the control condition (37.0±0.5^oC), but this difference was reduced to a non-significant 0.4^oC throughout exercise. Mean skin temperature was significantly lower in the pre-cooling (30.7±2.3^oC) than in the control condition (32.5±1.6^oC) throughout exercise. Heart rate during submaximal exercise was similar between the two conditions, although peak heart rate after the Wingate sprints was significantly lower in the pre-cooling condition. With pre-cooling, mean peak power (909±161?W) and mean overall power output (797±154?W) were similar to those in the control condition (peak 921±163?W, mean 806±156?W), with no differences in the subjective ratings of perceived exertion. These results suggest that upper-body pre-cooling does not provide any benefit to intermittent sprinting exercise in a moderate environment.     

Markers of isometric training intensity and reductions in resting blood pressure

Abstract

In this study, we examined the correlations between selected markers of isometric training intensity and subsequent reductions in resting blood pressure. Thirteen participants performed a discontinuous incremental isometric exercise test to volitional exhaustion at which point mean torque for the final 2-min stage (2min-torque_peak) and peak heart rate peak (HR_peak) were identified. Also, during 4 weeks of training (3 sessions per week, comprising 4 × 2 min bilateral leg isometric exercise at 95% HR_peak), heart rate (HR_train), torque (Torque_train), and changes in EMG amplitude (ΔEMG_amp) and frequency (ΔEMG_freq) were determined. The markers of training intensity were: Torque_train relative to the 2min-torque_peak (%2min-torque_peak), EMG relative to EMG_peak (%EMG_peak), HR_train ΔEMG_amp, ΔEMG_freq, and %MVC. Mean systolic (?4.9 mmHg) and arterial blood pressure (?2.7mmHg) reductions correlated with %2min-torque_peak (r = ?0.65, P = 0.02 and r = ?0.59, P = 0.03), ΔEMG_amp (r = 0.66, P = 0.01 and r = 0.59, P = 0.03), ΔEMG_freq (r = ?0.67, P = 0.01 and r = ?0.64, P = 0.02), and %EMG_peak (systolic blood pressure only; r = ?0.63, P = 0.02). These markers best reflect the association between isometric training intensity and reduction in resting blood pressure observed after bilateral leg isometric exercise training.     

No effect of glutamine supplementation and hyperoxia on oxidative metabolism and performance during high-intensity exercise

Abstract

Glutamine enhances the exercise-induced expansion of the tricarboxylic acid intermediate pool. The aim of the present study was to determine whether oral glutamine, alone or in combination with hyperoxia, influenced oxidative metabolism and cycle time-trial performance. Eight participants consumed either placebo or 0.125 g · kg body mass^?1 of glutamine in 5 ml · kg body mass^?1 placebo 1 h before exercise in normoxic (control and glutamine respectively) or hyperoxic (FiO₂ = 50%; hyperoxia and hyperoxia + glutamine respectively) conditions. Participants then cycled for 6 min at 70% maximal oxygen uptake ([Vdot]O_2max) immediately before completing a brief high-intensity time-trial (～4 min) during which a pre-determined volume of work was completed as fast as possible. The increment in pulmonary oxygen uptake during the performance test (Δ[Vdot]O_2max, P = 0.02) and exercise performance (control: 243 s, s _x  = 7; glutamine: 242 s, s _x  = 3; hyperoxia: 231 s, s _x  = 3; hyperoxia + glutamine: 228 s, s _x  = 5; P < 0.01) were significantly improved in hyperoxic conditions. There was some evidence that glutamine ingestion increased Δ[Vdot]O_2max in normoxia, but not hyperoxia (interaction drink/FiO₂, P = 0.04), but there was no main effect or impact on performance. Overall, the data show no effect of glutamine ingestion either alone or in combination with hyperoxia, and thus no limiting effect of the tricarboxylic acid intermediate pool size, on oxidative metabolism and performance during maximal exercise.     

Temporal Aspects of the VO2 Response at the Power Output Associated with VO2peak in Well Trained Cyclists—Implications for Interval Training Prescription

Abstract

The power output achieved at peak oxygen consumption (VO ₂Peak) and the time this power can be maintained (i. e., Tmax) have been used in prescribing high-intensity interval training. In this context, the present study examined temporal aspects of the VO₂ response to exercise at the cycling power that output well trained cyclists achieve their VO ₂peak (i. e., Pmax). Following a progressive exercise test to determine VO ₂peak, 43 well trained male cyclists (M age = 25 years, SD = 6; M mass = 75 kg, SD = 7; M VO₂ peak = 64.8 ml-kg¹ min^?1, SD = 5.2) performed two Tmax tests 1 week apart. Values expressed for each participant are means and standard deviations of these two tests. Participants achieved a mean VO ₂peak during the Tmax test after 176 s (SD = 40; M = 74% of Tmax, SD = 12) and maintained it for 66 s (SD = 39; M = 26% of Tmax, SD = 12). Additionally, they obtained mean 95% of VO ₂peak after 147 s (SD = 31; M = 62% of Tmax, SD = 8) and maintained it for 95 s (SD = 38; M = 38 % of Tmax, SD = 8). These results suggest that 60–70 % of Tmax is an appropriate exercise duration for a population of well trained cyclists to attain VO ₂peak during exercise at Pmax. However, due to intraparticipant variability in the temporal aspects of the VO₂ response to exercise at Pmax, future research is needed to examine whether individual high-intensity interval training programs for well trained endurance athletes might best be prescribed according to an athlete's individual VO₂ response to exercise at Pmax.     

Errors in the estimation of hydration status from changes in body mass

Abstract

Hydration status is not easily measured, but acute changes in hydration status are often estimated from body mass change. Changes in body mass are also often used as a proxy measure for sweat losses. There are, however, several sources of error that may give rise to misleading results, and our aim in this paper is to quantify these potential errors. Respiratory water losses can be substantial during hard work in dry environments. Mass loss also results from substrate oxidation, but this generates water of oxidation which is added to the body water pool, thus dissociating changes in body mass and hydration status: fat oxidation actually results in a net gain in body mass as the mass of carbon dioxide generated is less than the mass of oxygen consumed. Water stored with muscle glycogen is presumed to be made available as endogenous carbohydrate stores are oxidized. Fluid ingestion and sweat loss complicate the picture by altering body water distribution. Loss of hypotonic sweat results in increased osmolality of body fluids. Urine and faecal losses can be measured easily, but changes in the water content of the bladder and the gastrointestinal tract cannot. Body mass change is not always a reliable measure of changes in hydration status and substantial loss of mass may occur without an effective net negative fluid balance.     

Effects of audiovisual stimuli on psychological and psychophysiological responses during exercise in adults with obesity

The present experiment sought to further understanding of the effects of personalised audiovisual stimuli on psychological and psychophysiological responses during exercise in adults with obesity. Twenty-four participants (M_age = 28.3, SD = 5.5 years; M_BMI = 32.2, SD = 2.4) engaged in self-paced exercises on a recumbent cycle ergometer and three conditions (sensory stimulation [ST], sensory deprivation [DE], and control [CO]) were administered. Perceptual (attentional focus and perceived exertion), affective (affective state and perceived activation), and psychophysiological (heart rate variability) parameters were monitored throughout the exercise bouts. A one-way repeated measures analysis of variance was used to compare self-reported and psychophysiological variables (main and interaction effects [5 Timepoints × 3 Conditions]). The results indicate that ST increased the use of dissociative thoughts throughout the exercise session (η_p² = .19), ameliorated fatigue-related symptoms (η_p² = .15) and elicited more positive affective responses (η_p² = .12) than CO and DE. Accordingly, personally-compiled videos are highly effective in ameliorating exertional responses and enhancing affective valence during self-paced exercise in adults with obesity. Audiovisual stimuli could be used during the most critical periods of the exercise regimen (e.g., first training sessions) when individuals with obesity are more likely to focus on fatigue-related sensations.