Design and development of a new facility for teaching and research in clinical anatomy

This article discusses factors in the design, commissioning, project management, and intellectual property protection of developments within a new clinical anatomy facility in the United Kingdom. The project was aimed at creating cost‐effective facilities that would address widespread concerns over anatomy teaching, and support other activities central to the university mission–namely research and community interaction. The new facilities comprise an engaging learning environment and were designed to support a range of pedagogies appropriate to the needs of healthcare professionals at different stages of their careers. Specific innovations include integrated workstations each comprising of a dissection table, with removable top sections, an overhead operating light, and ceiling‐mounted camera. The tables incorporate waterproof touch‐screen monitors to display images from the camera, an endoscope or a database of images, videos, and tutorials. The screens work independently so that instructors can run different teaching sessions simultaneously and students can progress at different speeds to suit themselves. Further, database access is provided from within an integrated anatomy and pathology museum and display units dedicated to the correlation of cross‐sectional anatomy with medical imaging. A new functional neuroanatomy modeling system, called the BrainTower®, has been developed to aid integration of anatomy with physiology and clinical neurology. Many aspects of the new facility are reproduced within a Mobile Teaching Unit, which can be driven to hospitals, colleges, and schools to provide appropriate work‐based education and community interaction. Anat Sci Ed 2:34–40, 2009. © 2009 American Association of Anatomists.     

Urea production during prolonged swimming

Male interscholastic swimmers (n = 8) completed a 4572 m training swim in 62 ±1.1 min (x ± s.e.) with terminal heart rate and blood lactate of 152 ± 6 beats min^‐1 and 6.9±0.89 mM, respectively. Sweat rate (0.48±0.0951. h^‐1) was lower than similar intensity cycling (1.5±0.13 1. h^‐1) or running (1.1 ± 0.14 l.h^‐1). Post‐swim serum urea N (11.6±0.71 mM) was elevated (P<0.05) vs pre‐swim (4.6±0.39 mM). Post‐swim urine volume (860±75 ml 24 h^‐1) was reduced (P<0.07) and resulted in an elevated (P<0.05), but delayed (24–84 h), post‐exercise urea N excretion. Although the reduced urine and sweat production during the swim undoubtedly contributed to the elevated serum urea, there must be another explanation because together they could only account for 38% of the observed increase. On the basis of the magnitude of serum urea increase, it appears that the swim caused an increase in urea production (amino acid oxidation). The failure to observe larger increases in urinary urea during recovery indicates that either urea excretion following exercise continues for prolonged periods of time (>48 h) or another significant mode of nitrogen excretion exists.     

The effect of marathon running on carnitine metabolism and on some aspects of muscle mitochondrial activities and antioxidant mechanisms

Carnitine is an essential co‐factor in the catabolism of fats as an energy source. The primary purpose of this study was to investigate the effect of running a marathon on the metabolism of carnitine by endurance‐trained athletes, and to evaluate the effect of carnitine administration on the performance of such exercise. The effects of marathon running on mitochondrial enzymes and cellular anti‐oxidants were also examined to assess whether the expression of these activities is altered by exercise. Subjects were 10 experienced male marathon runners aged between 19 and 25 years. Running a marathon caused a fall in the plasma content of unesterified carnitine (37%) and an increase in the level of acetylcarnitine present (288%). Loading of the athletes with L‐carnitine for 10 days before running a marathon abolished the exercise‐induced fall in plasma‐free carnitine (P<0.05) whilst amplifying the production of acetylcarnitine (P<0.05). Carnitine loading of the athletes studied made no detectable improvement in performance of the marathon (P > 0.05). Cytochrome oxidase, succinate cytochrome C reductase and Superoxide dismutase activities present in skeletal muscle were unaltered by marathon running. However, such exercise caused a large increase in the tissue content of oxidized glutathione (189%) at the expense of reduced glutathione (–18%).     

Relationship between achievement goal orientations and the perceived purposes of playing rugby union for professional and amateur players

The recent professionalization of rugby union makes it an excellent achievement context in which to examine the relationship between achievement goal orientations and the perceived purposes of sport as a function of competitive standard. During the 1996–97 season, 73 professional and 106 amateur rugby players in England completed a series of questionnaires assessing their achievement goal orientations, beliefs about the purposes of rugby and demographic information. The results of a canonical correlation analysis revealed a conceptually coherent relationship between achievement goal orientations and purposes of rugby. Specifically, a high ego/ moderate task orientation was positively related to fitness, aggression and financial remuneration as significant purposes of rugby. Professional players scored higher on those purposes of rugby related to aggression, financial remuneration and fitness, but lower on sportspersonship than amateur players. Professional players also reported higher task and ego goal orientations than amateur players. The findings are discussed in terms of the differences in lifestyle and motivation of professional and amateur rugby union players.     

Talent identification and development in soccer

In this review, we attempt to integrate the main research findings concerned with talent identification and development in soccer. Research approaches in anthropometry, physiology, psychology and sociology are considered and, where possible, integrated. Although some progress has been made in identifying correlates of playing success, it appears that no unique characteristics can be isolated with confidence. Both biological and behavioural scientists have indicated a strong genetic component in performance of sports such as soccer; nevertheless, the influence of systematic training and development programmes should not be underestimated. Weconclude that the sport and exercise sciences have an important support role in the processes of identifying, monitoring and nurturing talented soccer players towards realizing their potential.     

Exercise does not necessarily influence the phase of the circadian rhythm in temperature in healthy humans

Seventeen healthy adults exercised for 30 min (70% V O 2max ), at different times of the day and night throughout a 24 h period. The effect on the phase of core temperature rhythm was assessed by comparing the rhythms on the day immediately before and immediately after the day of exercise with the participants living normally on these 2 days. Assessments were made upon data that had been corrected for the thermogenic effects of sleep and activity. When exercise was performed in the period between 4 h before and 1 h after the temperature minimum, there was a phase delay of 1.03 - 0.78 h (mean - s ; n = 6); when performed between 3 and 8 h after the temperature minimum, there was a phase advance of 1.07 - 1.23 h ( n = 9). Both means were significantly different from zero (Student's one-sample t -tests: P ? 0.05). Performed at other times, exercise had no significant effect on the phase of the temperature rhythm. The phase changes were not significantly different from those previously observed by us in sedentary individuals exposed to domestic lighting. We conclude that the amount of physical activity has very little effect on the body clock.     

Agreement between Polar and SRM mobile ergometer systems during laboratory-based high-intensity,intermittent cycling activity

Abstract

The purpose of this study was to assess the agreement between two mobile cycle ergometer systems for recording high-intensity, intermittent power output. Twelve trained male cyclists (age 31.4 ± 9.8 years) performed a single 3 min intermittent cycle test consisting of 12 all-out efforts, separated by periods of passive recovery ranging from 5 to 15 s. Power output was recorded using a Polar S710 heart rate monitor and power sensor kit and an SRM Powercrank system for each test. The SRM used torque and angular velocity to calculate power, while the S710 used chain speed and vibration to calculate power. Significant differences (P < 0.05) in power were found at 8 of the 12 efforts. A significant difference (P = 0.001) was also found when power was averaged over all 12 intervals. Mean power was 556 ± 102 W and 446 ± 61 W for the SRM and S710 respectively. The S710 underestimated power by an average of 23% with random errors of ?/÷ 24% when compared with the SRM. Random errors ranged from 36% to 141% with a median of 51%. The results indicate there was little agreement between the two systems and that the Polar S710 did not provide a valid measure of power during intermittent cycling activity when compared with the SRM. Power recorded by the S710 system was influenced greatly by chain vibration and sampling rates.