软盘“不死”,只是“凋零”

鸡肋!鸡肋!食之无味,弃之可惜!用鸡肋来形容被时代潮流所淘汰的电脑软盘,似乎十分贴切恰当。不过与鸡肋相比,旧软盘的味道更加富有变化性,如果你很用心地吃它,它或许就会变成美味佳肴。虽然随着U盘、移动硬盘等便携性、大容量存储工具的出现,电脑软盘(A盘)淡出大家的视野是一种必然趋势,但是看着自己曾经用过的那么一大叠软盘,真的是欲扔又止。相信同学们也有着同样的感受,本期我爱DIY就将为大家介绍电脑软盘再利用的诸多办法。记得美国将军麦克阿瑟曾经说过老兵不死,只是凋零,如果你是一个好的主人,你的软盘也不会突然死亡,而会在发挥余热的过程中慢慢凋零。     

《碟中谍》中的高科技

人皮面具、口香糖炸弹、传达完任务后5秒钟自动销毁(xiao hui)的录音机……这些《碟(die)中谍(die)》系列中酷炫(xuan)的高科技设备一定给你留下深刻印象,而磁(ci)力攀(pan)爬手套、会障(zhang)眼法的屏幕、前方挡风玻璃(bo li)有地图显示的汽车……这些让人目不暇(xia)接的《碟中谍4》中的新奇玩意,更让人意想不到。     

“野象谷”的主人们

野象谷坐落在西双版纳景洪市勐养镇以北一个叫三岔河的地方,因野象经常出没而得名。这里河谷纵横,森林茂密,一片热带雨林风光,在这里生长着亚洲野象、野牛、绿孔雀、猕     

悬疑电话

前不久,编辑部传言,过几天要来一个姓高的新同事,绝对的创意型人才,据说智商160不打折,虽然刚刚大学毕业,但是已经有很多家公司对他虎视眈眈了。众编辑听闻后都对这个神乎其神的天才人物相当好奇,眼巴巴地等着他的到来。终于有一天,这位高姓同事驾到编辑部。大家列队欢迎。只见一个袖珍的小男生从远处走了过来。此人     

Induction of experimental acute ulcerative colitis in rats by administration of dextran sulfate sodium at low concentration followed by intracolonic administration of 30% ethanol

Several models of experimental ulcerative colitis have been reported previously. However, none of these models showed the optimum characteristics. Although dextran sulfate sodium-induced colitis results in inflammation resembling ulcera-tive colitis, an obvious obstacle is that dextran sulfate sodium is very expensive. The aim of this study was to develop an inex-pensive model of colitis in rats. Sprague-Dawley rats were treated with 2% dextran sulfate sodium in drinking water for 3 d fol-lowed by an intracolonic administration of 30% ethanol. The administration of 2% dextran sulfate sodium followed by 30% ethanol induced significant weight loss, diarrhea and hematochezia in rats. Severe ulceration and inflammation of the distal part of rat colon were developed rapidly. Histological examination showed increased infiltration of polymorphonuclear leukocytes, lymphocytes and existence of cryptic abscesses and dysplasia. The model induced by dextran sulfate sodium at lower concentra-tion followed by 30% ethanol is characterized by a clinical course, localization of the lesions and histopathological features similar to human ulcerative colitis and fulfills the criteria set out at the beginning of this study.     

Colorectal cancer cell growth inhibition by linoleic acid is related to fatty acid composition changes

Polyunsaturated fatty acids (PUFAs) possess anti-cancer action both in vitro and in vivo. In the present study, we detected cell viability with methyl thiazolyl tetrazolium (MTT) assay and cell membrane permeability with propidium iodide (PI) fluorescence dyeing, and calculated cell membrane fluidity change as fluorescence anisotropy. Fatty acid content in cells was measured by gas chromatography/mass spectroscopy (GC/MS), and the relationship between fatty acid composition and cell viability was studied. We observed that n-6 PUFA linoleic acid (LA) inhibited tumor cell growth at high concentrations (≥300 μmol/L), while low concentrations (100–200 μmol/L) seemed to promote cell proliferation. Analyses of cell membrane permeability, cell membrane fluidity, and cell fatty acid composition suggested that the anti-cancer action of LA could be related to changes in the ratio of n-6 to n-3 PUFAs. We observed that pre-incubation of cancer cells with 100 μmol/L LA for 24 h enhanced cell sensitivity to the cytotoxic action of LA, whereas undifferentiated cell line LoVo seemed to have a distinct path in LA-induced death. These results showed that one of the mechanisms by which supplementation of LA induces cancer cell death could be altering the ratio of n-6/n-3 PUFAs, and this may be related to cell differentiation status.