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121.
122.
运用中医理论结合现代医学思想,对优秀冰雪运动员“运动性疲劳”症候进行调查、分析,制订出一套“运动性疲劳”的中医分型方法,并提出各型疲劳症候的诊断标准,以及“运动性疲劳”、“虚劳”与“脏腑虚劳”的鉴别诊断。 相似文献
123.
Abstract This case study examines the experiences of a student who was excluded twice from a mainstream school while preparing for GCSE examinations. The authors are the student and his aunt. The exclusions were for an indefinite period and were triggered as a result of the school's inability to respond adequately to recurring epileptic seizures. The student, who has Asperger syndrome and a statement of special educational needs, had not broken any disciplinary code. Teachers' understanding of the label ‘special educational needs’ is questioned. The SEN and Disability Act 2001 has far-reaching implications for school ethos and culture. Schools' failure to anticipate the needs of students with disabilities or SEN may well lead to unlawful discrimination. Inclusive schools will need to recognize that in meeting students' individual needs the institution itself may need to change. Barriers to genuine parent partnership in education must be overcome, and children's participation rights, as confirmed in the UN Convention on the Rights of the Child, must be respected. 相似文献
124.
On the basis of their cognitive abilities, children with Asperger syndrome are attractive candidates for inclusive education and, in Australia, most are in integrated settings. However, social interaction between children with Asperger syndrome and their peers remains problematic, with the children with Asperger syndrome often being left alone despite being among or near classmates. For classroom teachers, effective interventions in terms of class management are critical to facilitate positive social interaction between these two groups of children, and case studies can tell us a great deal about what works. In practice, a combination of intervention techniques may be best, in case a single one is ineffective, and in order to take advantage of potential synergies. In this article, Serene Choi, of the School of Education at the University of Newcastle (Australia) and Timo Nieminen, of the Centre for Biophotonics and Laser Science in the Department of Physics at the University of Queensland, report a naturalistic multiple-component intervention used to develop the social skills of a boy with Asperger syndrome in a mainstream school in Australia. This combined intervention, making use of social stories, simple peer modelling and individual lessons, appears to be a practical teaching method for inclusive education. 相似文献
125.
运动性脾气虚证研究现状与展望 总被引:1,自引:0,他引:1
采用献资料法,综述了近年来有关运动性脾气虚证的诊断标准、动物实验和临床研究现状,分析了目前研究中存在的问题,提出今后的研究方向,为运动性脾气虚证本质的系统研究提供了参考。 相似文献
126.
本文探讨了过度训练引起的症状,即过度训练综合症(简称OTS)的外部表现及内部机制,包括OTS与神经系统,内分泌系统及免疫系统的关系,提出了预防及判断OTS的生理指标及消除OTS的具体方法。 相似文献
127.
运动干预代谢综合征机制研究进展 总被引:6,自引:0,他引:6
运动可通过提高胰岛素敏感性及葡萄糖的转运速度,促进骨骼肌利用葡萄糖而降低血糖;减轻体重,改善内脏脂肪堆积,降低血清低密度脂蛋白水平,促进胆固醇的逆向转运;改善血管内皮功能,降低血压,干预代谢综合征的发生与发展,减轻糖尿病、肥胖、高血压、血脂异常等代谢综合征的临床表现,起到预防和辅助药物治疗的作用。 相似文献
128.
Munchausen syndrome by proxy: A family affair 总被引:1,自引:0,他引:1
Munchausen syndrome by proxy is an unusual form of child abuse: a child presents with an illness that has been factitiously produced by a parent, typically the mother. A case of chronic illicit insulin administration to a one-year-old girl is described. Despite temporary separation of the child from the mother and long-term psychiatric intervention, factitious illnesses continued, including urine specimen contamination, laxative-induced diarrhea, suspected bladder catheterization, and suspected poisoning. Retrospective review of the medical records of the mother and two siblings demonstrated previously unrecognized evidence of factitious illnesses. The medical records contained evidence of 30 separate episodes of suspected or documented factitious illness in these four members of the same family. This unique family illustrates the significant morbidity of Munchausen syndrome by proxy and a poor response to psychiatric treatment. 相似文献
129.
Liang Liu Bei-ni Liu Shuo Chen Miao Wang Yang Liu Yan-li Zhang Shu-kun Yao 《Journal of Zhejiang University. Science. B》2014,15(10):907-914
The pathophysiology of irritable bowel syndrome (IBS) is complex and not fully understood, so the aim of this study was to evaluate whether visceral and somatic hypersensitivity, autonomic cardiovascular dysfunction, and low-grade inflammation of the gut wall are associated with diarrhea-predominant IBS (D-IBS). Sixty-two patients with D-IBS and 20 control subjects participated in the study. Using the ascending method of limits (AML) protocol, we demonstrated that D-IBS patients had significantly lower sensory thresholds compared with healthy controls (P<0.001). Using diverse methods, especially the ischemic sensitivity test, for the first time in China, we confirmed that D-IBS patients have somatic hypersensitivity. They had a significantly higher systolic blood pressure and heart rate after a cold stimulus, indicative of autonomic cardiovascular dysfunction. Compared with the control group, D-IBS patients had a significantly higher level of calprotectin (P<0.001). We also found significant correlations between visceral and somatic hypersensitivity, visceral hypersensitivity and autonomic cardiovascular dysfunction, and somatic hypersensitivity and autonomic cardiovascular dysfunction. Our findings may provide valuable suggestions for the treatment of D-IBS. 相似文献
130.
Yan LI Yu-jin QU Xue-mei ZHONG Yan-yan CAO Li-min JIN Jin-li BAI Xin MA Yu-wei JIN Hong WANG Yan-ling ZHANG Fang SONG 《Journal of Zhejiang University. Science. B》2014,15(5):474-481
Crigler-Najjar syndrome type I (CN-I) is the most severe type of hereditary unconjugated hyperbilirubinemia. It is caused by homozygous or compound heterozygous mutations of the UDP-glycuronosyltransferase gene (UGT1A1) on chromosome 2q37. Two patients clinically diagnosed with CN-I were examined in this paper. We sequenced five exons and their flanking sequences, specifically the promoter region of UGT1A1, of the two patients and their parents. Quantitative real-time polymerase chain reaction (qRT-PCR) was used to determine the UGT1A1 gene copy number of one patient. In patient A, two mutations, c.239_245delCTGTGCC (p.Pro80HisfsX6; had not been reported previously) and c.1156G>T (p.Val386Phe), were identified. In patient B, we found that this patient had lost heterozygosity of the UGT1A1 gene by inheriting a deletion of one allele, and had a novel mutation c.1253delT (p.Met418ArgfsX5) in the other allele. In summary, we detected three UGT1A1 mutations in two CN-I patients: c.239_245delCTGTGCC (p.Pro80HisfsX6), c.1253delT (p.Met418ArgfsX5), and c.1156G>T (p.Val386Phe). The former two mutations are pathogenic; however, the pathogenic mechanism of c.1156G>T (p.Val386Phe) is unknown. 相似文献